Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Research Project (R01)
Project #
3R01CA077465-01S1
Application #
2870265
Study Section
Radiation Study Section (RAD)
Program Officer
Mahoney, Francis J
Project Start
1998-04-01
Project End
2001-03-31
Budget Start
1998-04-01
Budget End
1999-03-31
Support Year
1
Fiscal Year
1998
Total Cost
Indirect Cost
Name
Dana-Farber Cancer Institute
Department
Type
DUNS #
149617367
City
Boston
State
MA
Country
United States
Zip Code
02215
Calderwood, Stuart K; Gong, Jianlin (2012) Molecular chaperones in mammary cancer growth and breast tumor therapy. J Cell Biochem 113:1096-103
Zhang, Yue; Chou, Shiuh-Dih; Murshid, Ayesha et al. (2011) The role of heat shock factors in stress-induced transcription. Methods Mol Biol 787:21-32
Murshid, Ayesha; Gong, Jianlin; Stevenson, Mary Ann et al. (2011) Heat shock proteins and cancer vaccines: developments in the past decade and chaperoning in the decade to come. Expert Rev Vaccines 10:1553-68
Murshid, Ayesha; Chou, Shiuh-Dih; Prince, Thomas et al. (2010) Protein kinase A binds and activates heat shock factor 1. PLoS One 5:e13830
Calderwood, Stuart K (2010) Heat shock proteins in breast cancer progression--a suitable case for treatment? Int J Hyperthermia 26:681-5
Calderwood, Stuart K; Murshid, Ayesha; Prince, Thomas (2009) The shock of aging: molecular chaperones and the heat shock response in longevity and aging--a mini-review. Gerontology 55:550-8
Gong, Jianlin; Zhu, Bangmin; Murshid, Ayesha et al. (2009) T cell activation by heat shock protein 70 vaccine requires TLR signaling and scavenger receptor expressed by endothelial cells-1. J Immunol 183:3092-8
Gray Jr, Phillip J; Prince, Thomas; Cheng, Jinrong et al. (2008) Targeting the oncogene and kinome chaperone CDC37. Nat Rev Cancer 8:491-5
Khaleque, M A; Bharti, A; Gong, J et al. (2008) Heat shock factor 1 represses estrogen-dependent transcription through association with MTA1. Oncogene 27:1886-93
Calderwood, Stuart K (2005) Regulatory interfaces between the stress protein response and other gene expression programs in the cell. Methods 35:139-48

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