Agency
National Institute of Health (NIH)
Institute
National Institute of Dental & Craniofacial Research (NIDCR)
Type
Research Project (R01)
Project #
1R01DE007378-01
Application #
3221037
Study Section
Oral Biology and Medicine Study Section (OBM)
Project Start
1985-09-01
Project End
1988-08-31
Budget Start
1985-09-01
Budget End
1986-08-31
Support Year
1
Fiscal Year
1985
Total Cost
Indirect Cost
Name
Forsyth Institute
Department
Type
DUNS #
City
Cambridge
State
MA
Country
United States
Zip Code
Battaglino, R A; Pham, L; Morse, L R et al. (2008) NHA-oc/NHA2: a mitochondrial cation-proton antiporter selectively expressed in osteoclasts. Bone 42:180-92
Morse, L; Teng, Y D; Pham, L et al. (2008) Spinal cord injury causes rapid osteoclastic resorption and growth plate abnormalities in growing rats (SCI-induced bone loss in growing rats). Osteoporos Int 19:645-52
Pham, L; Purcell, P; Morse, L et al. (2007) Expression analysis of nha-oc/NHA2: a novel gene selectively expressed in osteoclasts. Gene Expr Patterns 7:846-51
Schulze-Spate, Ulrike; Battaglino, Ricardo; Fu, Jia et al. (2007) Brn3 transcription factors control terminal osteoclastogenesis. J Cell Biochem 102:1-12
Battaglino, R; Vokes, M; Schulze-Spate, U et al. (2007) Fluoxetine treatment increases trabecular bone formation in mice. J Cell Biochem 100:1387-94
Keles, Ahmet; Grunes, Brandon; Difuria, Catherine et al. (2007) Inhibition of tooth movement by osteoprotegerin vs. pamidronate under conditions of constant orthodontic force. Eur J Oral Sci 115:131-6
Okamatsu, Yoshimasa; Kim, David; Battaglino, Ricardo et al. (2004) MIP-1 gamma promotes receptor-activator-of-NF-kappa-B-ligand-induced osteoclast formation and survival. J Immunol 173:2084-90
Battaglino, R; Kim, D; Fu, J et al. (2002) c-myc is required for osteoclast differentiation. J Bone Miner Res 17:763-73
Deng, W; Stashenko, P; Chen, W et al. (2001) Characterization of mouse Atp6i gene, the gene promoter, and the gene expression. J Bone Miner Res 16:1136-46
Li, Y P; Chen, W; Liang, Y et al. (1999) Atp6i-deficient mice exhibit severe osteopetrosis due to loss of osteoclast-mediated extracellular acidification. Nat Genet 23:447-51

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