Agency
National Institute of Health (NIH)
Institute
National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
Type
Research Project (R01)
Project #
3R01DK054740-05S1
Application #
6802564
Study Section
Pathology A Study Section (PTHA)
Program Officer
Wilder, Elizabeth L
Project Start
1999-01-15
Project End
2007-03-31
Budget Start
2003-08-05
Budget End
2004-03-31
Support Year
5
Fiscal Year
2003
Total Cost
$99,210
Indirect Cost
Name
University of Michigan Ann Arbor
Department
Pathology
Type
Schools of Medicine
DUNS #
073133571
City
Ann Arbor
State
MI
Country
United States
Zip Code
48109
Sun, Yuqing; Zhou, Bo; Mao, Fengbiao et al. (2018) HOXA9 Reprograms the Enhancer Landscape to Promote Leukemogenesis. Cancer Cell 34:643-658.e5
Ihermann-Hella, Anneliis; Hirashima, Tsuyoshi; Kupari, Jussi et al. (2018) Dynamic MAPK/ERK Activity Sustains Nephron Progenitors through Niche Regulation and Primes Precursors for Differentiation. Stem Cell Reports 11:912-928
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Soofi, Abdul; Wolf, Katherine I; Ranghini, Egon J et al. (2016) The kielin/chordin-like protein KCP attenuates nonalcoholic fatty liver disease in mice. Am J Physiol Gastrointest Liver Physiol 311:G587-G598
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Abraham, Saji; Paknikar, Raghavendra; Bhumbra, Samina et al. (2015) The Groucho-associated phosphatase PPM1B displaces Pax transactivation domain interacting protein (PTIP) to switch the transcription factor Pax2 from a transcriptional activator to a repressor. J Biol Chem 290:7185-94
Ranghini, Egon J; Dressler, Gregory R (2015) Evidence for intermediate mesoderm and kidney progenitor cell specification by Pax2 and PTIP dependent mechanisms. Dev Biol 399:296-305

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