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Nenov, N I; Crumb Jr, W J; Pigott, J D et al. (1998) Quinidine interactions with human atrial potassium channels: developmental aspects. Circ Res 83:1224-31
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Roca, T P; Pigott, J D; Clarkson, C W et al. (1996) L-type calcium current in pediatric and adult human atrial myocytes: evidence for developmental changes in channel inactivation. Pediatr Res 40:462-8
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Clarkson, C W; Xu, Y Q; Chang, C et al. (1996) Analysis of the ionic basis for cocaine's biphasic effect on action potential duration in guinea-pig ventricular myocytes. J Mol Cell Cardiol 28:667-78
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Crumb Jr, W J; Pigott, J D; Clarkson, C W (1995) Comparison of Ito in young and adult human atrial myocytes: evidence for developmental changes. Am J Physiol 268:H1335-42
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Crumb Jr, W J; Pigott, J D; Clarkson, C W (1995) Description of a nonselective cation current in human atrium. Circ Res 77:950-6
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Crumb Jr, W J; Clarkson, C W (1995) The pH dependence of cocaine interaction with cardiac sodium channels. J Pharmacol Exp Ther 274:1228-37
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Xu, Y Q; Crumb Jr, W J; Clarkson, C W (1994) Cocaethylene, a metabolite of cocaine and ethanol, is a potent blocker of cardiac sodium channels. J Pharmacol Exp Ther 271:319-25
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Clarkson, C W; Chang, C; Stolfi, A et al. (1993) Electrophysiological effects of high cocaine concentrations on intact canine heart. Evidence for modulation by both heart rate and autonomic nervous system. Circulation 87:950-62
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Crumb Jr, W J; Clarkson, C W (1992) Characterization of the sodium channel blocking properties of the major metabolites of cocaine in single cardiac myocytes. J Pharmacol Exp Ther 261:910-7
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Xu, Y Q; Pickoff, A S; Clarkson, C W (1992) Developmental changes in the effects of lidocaine on sodium channels in rat cardiac myocytes. J Pharmacol Exp Ther 262:670-6
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