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Silinsky, Eugene M (2013) Low-frequency neuromuscular depression is a consequence of a reduction in nerve terminal Ca2+ currents at mammalian motor nerve endings. Anesthesiology 119:326-34
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Searl, Timothy J; Silinsky, Eugene M (2012) Evidence for constitutively-active adenosine receptors at mammalian motor nerve endings. Eur J Pharmacol 685:38-41
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Searl, T J; Silinsky, E M (2012) Modulation of purinergic neuromuscular transmission by phorbol dibutyrate is independent of protein kinase C in murine urinary bladder. J Pharmacol Exp Ther 342:312-7
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Searl, Timothy J; Silinsky, Eugene M (2008) Mechanisms of neuromodulation as dissected using Sr2+ at motor nerve endings. J Neurophysiol 99:2779-88
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Silinsky, Eugene M (2008) Selective disruption of the mammalian secretory apparatus enhances or eliminates calcium current modulation in nerve endings. Proc Natl Acad Sci U S A 105:6427-32
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Searl, Timothy J; Silinsky, Eugene M (2005) Modulation of Ca(2+)-dependent and Ca(2+)-independent miniature endplate potentials by phorbol ester and adenosine in frog. Br J Pharmacol 145:954-62
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Silinsky, Eugene M (2005) Modulation of calcium currents is eliminated after cleavage of a strategic component of the mammalian secretory apparatus. J Physiol 566:681-8
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Silinsky, Eugene M (2004) Adenosine decreases both presynaptic calcium currents and neurotransmitter release at the mouse neuromuscular junction. J Physiol 558:389-401
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Searl, T J; Silinsky, E M (2003) Phorbol esters and adenosine affect the readily releasable neurotransmitter pool by different mechanisms at amphibian motor nerve endings. J Physiol 553:445-56
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Hirsh, Jody K; Searl, Timothy J; Silinsky, Eugene M (2002) Regulation by Rab3A of an endogenous modulator of neurotransmitter release at mouse motor nerve endings. J Physiol 545:337-43
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Showing the most recent 10 out of 35 publications
