Agency
National Institute of Health (NIH)
Institute
National Institute of Neurological Disorders and Stroke (NINDS)
Type
Research Project (R01)
Project #
5R01NS025987-05
Application #
3411581
Study Section
Neurological Sciences Subcommittee 1 (NLS)
Project Start
1988-12-01
Project End
1993-11-30
Budget Start
1991-12-01
Budget End
1992-11-30
Support Year
5
Fiscal Year
1992
Total Cost
Indirect Cost
Name
Tulane University
Department
Type
Schools of Medicine
DUNS #
City
New Orleans
State
LA
Country
United States
Zip Code
70118
Khodr, Christina E; Clark, Sara; Bokov, Alex F et al. (2010) Early postnatal administration of growth hormone increases tuberoinfundibular dopaminergic neuron numbers in Ames dwarf mice. Endocrinology 151:3277-85
Khodr, C E; Hurley, D L; Phelps, C J (2009) Prolactin induces tuberoinfundibular dopaminergic neurone differentiation in Snell dwarf mice if administered beginning at 3 days of age. J Neuroendocrinol 21:558-67
Khodr, Christina E; Clark, Sara M; Hurley, David L et al. (2008) Long-term, homologous prolactin, administered through ectopic pituitary grafts, induces hypothalamic dopamine neuron differentiation in adult Snell dwarf mice. Endocrinology 149:2010-8
Phelps, C J (2004) Postnatal regression of hypothalamic dopaminergic neurons in prolactin-deficient Snell dwarf mice. Endocrinology 145:5656-64
Phelps, Carol J; Romero, Mario I; Hurley, David L (2003) Prolactin replacement must be continuous and initiated prior to 21 d of age to maintain hypothalamic dopaminergic neurons in hypopituitary mice. Endocrine 20:139-48
Hurley, David L; Birch, Derin V; Almond, M Camille et al. (2003) Reduced hypothalamic neuropeptide Y expression in growth hormone- and prolactin-deficient Ames and Snell dwarf mice. Endocrinology 144:4783-9
Phelps, C J; Romero, M I; Hurley, D L (2003) Growth hormone-releasing hormone-producing and dopaminergic neurones in the mouse arcuate nucleus are independently regulated populations. J Neuroendocrinol 15:280-8
Wojtkiewicz, Patrick W; Phelps, Carol J; Hurley, David L (2002) Transcript abundance in mouse pituitaries with altered growth hormone expression quantified by reverse transcriptase polymerase chain reaction implicates transcription factor Zn-16 in gene regulation in vivo. Endocrine 18:67-74
Phelps, C J; Horseman, N D (2000) Prolactin gene disruption does not compromise differentiation of tuberoinfundibular dopaminergic neurons. Neuroendocrinology 72:10-Feb
VanderHeyden, T C; Wojtkiewicz, P W; Voss, T C et al. (2000) Mouse growth hormone transcription factor Zn-16: unique bipartite structure containing tandemly repeated zinc finger domains not reported in rat Zn-15. Mol Cell Endocrinol 159:89-98

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