Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of Neurological Disorders and Stroke (NINDS)
Type
Research Project (R01)
Project #
5R01NS032748-02
Application #
2271144
Study Section
Neurology B Subcommittee 2 (NEUB)
Project Start
1995-03-15
Project End
1998-02-28
Budget Start
1996-03-01
Budget End
1997-02-28
Support Year
2
Fiscal Year
1996
Total Cost
Indirect Cost

  Institution

Name
Florida State University
Department
Biology
Type
Schools of Arts and Sciences
DUNS #
020520466
City
Tallahassee
State
FL
Country
United States
Zip Code
32306

  Publications

Kim, Eun Young; Alvarez-Baron, Claudia P; Dryer, Stuart E (2009) Canonical transient receptor potential channel (TRPC)3 and TRPC6 associate with large-conductance Ca2+-activated K+ (BKCa) channels: role in BKCa trafficking to the surface of cultured podocytes. Mol Pharmacol 75:466-77
Jha, Smita; Dryer, Stuart E (2009) The beta1 subunit of Na+/K+-ATPase interacts with BKCa channels and affects their steady-state expression on the cell surface. FEBS Lett 583:3109-14
Kim, Eun Young; Choi, Kyoung-Jae; Dryer, Stuart E (2008) Nephrin binds to the COOH terminus of a large-conductance Ca2+-activated K+ channel isoform and regulates its expression on the cell surface. Am J Physiol Renal Physiol 295:F235-46
Zou, Shengwei; Jha, Smita; Kim, Eun Young et al. (2008) The beta 1 subunit of L-type voltage-gated Ca2+ channels independently binds to and inhibits the gating of large-conductance Ca2+-activated K+ channels. Mol Pharmacol 73:369-78
Zou, Shengwei; Jha, Smita; Kim, Eun Young et al. (2008) A novel actin-binding domain on Slo1 calcium-activated potassium channels is necessary for their expression in the plasma membrane. Mol Pharmacol 73:359-68
Kim, E Y; Ridgway, L D; Zou, S et al. (2007) Alternatively spliced C-terminal domains regulate the surface expression of large conductance calcium-activated potassium channels. Neuroscience 146:1652-61
Kim, Eun Young; Ridgway, Lon D; Dryer, Stuart E (2007) Interactions with filamin A stimulate surface expression of large-conductance Ca2+-activated K+ channels in the absence of direct actin binding. Mol Pharmacol 72:622-30
Kim, Eun Young; Zou, Shengwei; Ridgway, Lon D et al. (2007) Beta1-subunits increase surface expression of a large-conductance Ca2+-activated K+ channel isoform. J Neurophysiol 97:3508-16
Chae, Kwon-Seok; Oh, Kwang-Seok; Dryer, Stuart E (2005) Growth factors mobilize multiple pools of KCa channels in developing parasympathetic neurons: role of ADP-ribosylation factors and related proteins. J Neurophysiol 94:1597-605
Chae, Kwon-Seok; Dryer, Stuart E (2005) The p38 mitogen-activated protein kinase pathway negatively regulates Ca2+-activated K+ channel trafficking in developing parasympathetic neurons. J Neurochem 94:367-79

Showing the most recent 10 out of 30 publications