Abstract

The goals of this application for MBRS SCORE funding are to expand and strengthen Tuskegee University's long-standing commitment to biomedical research by: (1) Increasing the number of independent, self-reliant biomedical research scientists. (2) Increasing the number of research-related activities. (3) Coordinating the research activities between the Tuskegee University research community, support services and administration. The administrative and subproject components within this proposal will allow the development and support of biomedical research laboratories as well as provide essential support for research-related activities. It will be the job of the Program Director, through support of an Administrative Staff and consultations with an Advisory Committee, to directly advise and monitor the progress of all the MBRS investigators. Progress toward the stated goals will be assessed through the measurable objectives set for individual MBRS investigators, as well as the program as a whole.

  Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
Minority Biomedical Research Support - MBRS (S06)
Project #
5S06GM008091-32
Application #
6654429
Study Section
Minority Programs Review Committee (MPRC)
Program Officer
Carter, Anthony D
Project Start
1978-06-01
Project End
2006-08-31
Budget Start
2003-09-01
Budget End
2004-08-31
Support Year
32
Fiscal Year
2003
Total Cost
$229,998
Indirect Cost

  Institution

Name
Tuskegee University
Department
Biology
Type
Schools of Arts and Sciences
DUNS #
128214178
City
Tuskegee
State
AL
Country
United States
Zip Code
36088

  Publications

Mathews, Ensa; Braden, Tim D; Williams, Carol S et al. (2009) Mal-development of the penis and loss of fertility in male rats treated neonatally with female contraceptive 17alpha-ethinyl estradiol: a dose-response study and a comparative study with a known estrogenic teratogen diethylstilbestrol. Toxicol Sci 112:331-43
Goyal, H O; Braden, T D; Williams, C S et al. (2009) Estrogen-induced developmental disorders of the rat penis involve both estrogen receptor (ESR)- and androgen receptor (AR)-mediated pathways. Biol Reprod 81:507-16
Raboin, Shannon J; Reeve Jr, Joseph R; Cooper, Marvis S et al. (2008) Activation of submucosal but not myenteric plexus of the gastrointestinal tract accompanies reduction of food intake by camostat. Regul Pept 150:73-80
Cooper, Marvis S; Reeve Jr, Joseph R; Raboin, Shannon J et al. (2008) Cholecystokinin-58 and cholecystokinin-8 produce similar but not identical activations of myenteric plexus and dorsal vagal complex. Regul Pept 148:88-94
Cooper, Marvis S; Reeve Jr, Joseph R; Abdalla, Mohamed O et al. (2008) Cholecystokinin-33 is more effective than cholecystokinin-8 in inhibiting food intake and in stimulating the myenteric plexus and dorsal vagal complex. Brain Res 1205:27-35
Goyal, H O; Braden, T D; Williams, C S et al. (2007) Role of estrogen in induction of penile dysmorphogenesis: a review. Reproduction 134:199-208
Goyal, H O; Braden, T D; Cooke, P S et al. (2007) Estrogen receptor alpha mediates estrogen-inducible abnormalities in the developing penis. Reproduction 133:1057-67
Sullivan, Cherese N; Raboin, Shannon J; Gulley, Stephen et al. (2007) Endogenous cholecystokinin reduces food intake and increases Fos-like immunoreactivity in the dorsal vagal complex but not in the myenteric plexus by CCK1 receptor in the adult rat. Am J Physiol Regul Integr Comp Physiol 292:R1071-80
Raboin, Shannon J; Gulley, Stephen; Henley, Sheryce C et al. (2006) Atropine methyl nitrate increases myenteric but not dorsal vagal complex Fos-like immunoreactivity in the rat. Physiol Behav 88:448-52
Raboin, Shannon J; Gulley, Stephen; Henley, Sheryce C et al. (2006) Effect of sympathectomy and demedullation on increased myenteric and dorsal vagal complex Fos-like immunoreactivity by cholecystokinin-8. Regul Pept 134:141-8

Showing the most recent 10 out of 29 publications