The continuing long=-range objective of the MBRS Program at the University of Puerto Rico Medical Sciences Campus (MSC-MBRS) is targeted to achieve two principal goals; to help faculty develop a productive research project and to stimulate Hispanic students to choose a biomedical career. These goals, mutually interdependent, are pursued by facilitating the ongoing research projects and providing productive research environment ot stimulate enthusiastic students. Through the support of MBRS, investigators accumulate enough data to publish in peer-review journals and apply for independent research support. The Program focuses to encourage the participation of young investigators with the expectation that this support empowers them to compete on a more equal basis with researchers from more gifted institutions. Students from the Sch of Med Graduate Sch, the Sch of Pharm and undergraduates from the departments of Chemistry and Biology of the University of Puerto Rico, Rio Piedras Campus participate ina the Program.
We aim to encourage their long-term interest in biomedical research and careers by providing quality research experience. The student participants in the MSC-MBRS Program have achieved significant goals including acceptance on major institutions, such as Albert Einstein Sch of Med, University of Pittsburgh and Columbia University. Our application includes subproposls for 16 investigators of which three are associate investigators. The investigators are from the Schools of Medicine, and Pharmacy. The projects cover the areas of molecular biology, renal physiology, experimental therapeutics, toxicology, microbiology, parasite immunology, biochemistry, immunology and neurobiology.

Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
Minority Biomedical Research Support - MBRS (S06)
Project #
3S06GM008224-14S1
Application #
2873279
Study Section
Minority Programs Review Committee (MPRC)
Project Start
1985-05-01
Project End
2000-07-31
Budget Start
1998-08-01
Budget End
1999-07-31
Support Year
14
Fiscal Year
1998
Total Cost
Indirect Cost
Name
University of Puerto Rico Med Sciences
Department
Pharmacology
Type
Schools of Medicine
DUNS #
City
San Juan
State
PR
Country
United States
Zip Code
00936
Rijpma, Sanna R; van der Velden, Maarten; González-Pons, Maria et al. (2016) Multidrug ATP-binding cassette transporters are essential for hepatic development of Plasmodium sporozoites. Cell Microbiol 18:369-83
Padín-Irizarry, Vivian; Colón-Lorenzo, Emilee E; Vega-Rodríguez, Joel et al. (2016) Glutathione-deficient Plasmodium berghei parasites exhibit growth delay and nuclear DNA damage. Free Radic Biol Med 95:43-54
Jardón, Javier; Izquierdo, Natalio J; Renta, Jessica Y et al. (2016) Ocular Findings in Patients with the Hermansky-Pudlak Syndrome (Types 1 and 3). Ophthalmic Genet 37:89-94
Rivera-Peña, Bianca; Ruíz-Fullana, Francisco J; Vélez-Reyes, Germán L et al. (2016) HPV-16 infection modifies overall survival of Puerto Rican HNSCC patients. Infect Agent Cancer 11:47
Velásquez-Martínez, Maria C; Vázquez-Torres, Rafael; Rojas, Legier V et al. (2015) Alpha-1 adrenoreceptors modulate GABA release onto ventral tegmental area dopamine neurons. Neuropharmacology 88:110-21
Vega-Rodríguez, Joel; Pastrana-Mena, Rebecca; Crespo-Lladó, Keila N et al. (2015) Implications of Glutathione Levels in the Plasmodium berghei Response to Chloroquine and Artemisinin. PLoS One 10:e0128212
Zenón, Frances; Cantres-Rosario, Yisel; Adiga, Radhika et al. (2015) HIV-infected microglia mediate cathepsin B-induced neurotoxicity. J Neurovirol 21:544-58
Ortiz, A P; Unger, E R; Muñoz, C et al. (2014) Cross-sectional study of HPV-16 infection in a population-based subsample of Hispanic adults. BMJ Open 4:e004203
Rosas, Odrick R; Torrado, Aranza I; Santiago, Jose M et al. (2014) Long-term treatment with PP2 after spinal cord injury resulted in functional locomotor recovery and increased spared tissue. Neural Regen Res 9:2164-73
Mosquera, Laurivette; Colón, Jennifer M; Santiago, José M et al. (2014) Tamoxifen and estradiol improved locomotor function and increased spared tissue in rats after spinal cord injury: their antioxidant effect and role of estrogen receptor alpha. Brain Res 1561:11-22

Showing the most recent 10 out of 174 publications