This tri-institutional Post-Doctoral training program in cellular and molecular mechanisms of allergy has been funded by NIAID since July, 1994. The faculty members are from three neighboring institutes: University of California, San Diego (UCSD), La Jolla Institute for Allergy and Immunology (LIAI), and The Scripps Research Institute (TSRI). The trainees will have opportunities to be exposed to a wide range of Allergy research topics. All faculty members have well-established NIH funded research programs; their research backgrounds are diverse and encompass allergy, immunology, biochemistry, cell biology, and molecular biology. The trainees can be involved in the following research areas: 1) regulation of IgE production; 2) biology of inflammatory cells, including mast cells and esosinophils; 3) biology of T cells; 4) cell receptors critically involved in allergic reactions; 5) signal transduction; 6) inflammatory mediators/cytokines; 7) functions of epithelial cells; and 8) adhesion. The goals of the program are: a) to provide trainees with a basic understanding of the pathogenesis of allergic diseases; and b) to foster the development of investigative skills in trainees, in particular, applying molecular and cellular biological approaches to study mechanisms of diseases. The program is open to M.D.s and Ph.D.s interested in disease-oriented research and committed to a career in basic research in allergic inflammation. M.D. and Ph.D. trainees are expected to devote full time to research, and training will be supplemented by conferences, seminars, journal clubs and courses. M.D.s will have a half day clinic/week to maintain their clinical skills in allergy and immunology. Trainees will develop a solid background in the molecular and cellular mechanisms of allergic inflammation and become qualified to embark upon their careers as independent investigators in allergy research.
| Bhatia, Prerana; Chen, Meng; Christiansen, Sandra (2018) Marijuana and stoned fruit. Ann Allergy Asthma Immunol 120:536-537 |
| Karta, Maya R; Rosenthal, Peter S; Beppu, Andrew et al. (2018) ?2 integrins rather than ?1 integrins mediate Alternaria-induced group 2 innate lymphoid cell trafficking to the lung. J Allergy Clin Immunol 141:329-338.e12 |
| Chen, Meng; Kim, Alexander; Zuraw, Bruce et al. (2018) Mast cell disorders: Protean manifestations and treatment responses. Ann Allergy Asthma Immunol 121:128-130 |
| Piña, Francisco; Yagisawa, Fumi; Obara, Keisuke et al. (2018) Sphingolipids activate the endoplasmic reticulum stress surveillance pathway. J Cell Biol 217:495-505 |
| Miller, Marina; Vuong, Christine; Garcia, Meghan Farrell et al. (2018) Does reduced zona pellucida binding protein 2 (ZPBP2) expression on chromosome 17q21 protect against asthma? J Allergy Clin Immunol 142:706-709.e4 |
| Geng, Bob; Eastman, Jacqueline J; Mori, Karen et al. (2017) Utility of minor determinants for skin testing in inpatient penicillin allergy evaluation. Ann Allergy Asthma Immunol 119:258-261 |
| Chen, Meng; Land, Michael (2017) Baked milk and baked egg oral immunotherapy. Immunotherapy 9:1201-1204 |
| Song, Dae Jin; Miller, Marina; Beppu, Andrew et al. (2017) Rhinovirus Infection of ORMDL3 Transgenic Mice Is Associated with Reduced Rhinovirus Viral Load and Airway Inflammation. J Immunol 199:2215-2224 |
| Eastman, Jacqueline J; Kim, Alex S (2017) A patient with idiopathic angioedema presenting with superior vena cava syndrome and lymphedema. Ann Allergy Asthma Immunol 118:257-258 |
| Eastman, Jacqueline J; Cavagnero, Kellen J; Deconde, Adam S et al. (2017) Group 2 innate lymphoid cells are recruited to the nasal mucosa in patients with aspirin-exacerbated respiratory disease. J Allergy Clin Immunol 140:101-108.e3 |
Showing the most recent 10 out of 58 publications
