Description) The Developmental Biology Training Program (DBTP) at Duke University has the mission of training predoctoral students in developmental biology and developmental genetics at the highest level. The program provides a broad intellectual landscape and encourages students to learn aspects of developmental biology well beyond the boundaries of their narrow research topics. This new program grew out of two very successful and ongoing training programs at Duke University. The Cell and Molecular Biology Training Program and the Genetics Training Program have been the source of most students who enter Duke University and then find that developmental biology is attractive to them as a research area. Five years ago, with the number of students interested in developmental biology growing, the faculty in the basic sciences began to build the Developmental Biology Training Program. At the core of which is the excellent research environment in the basic sciences of the Medical School and the biological sciences at the College. Eight departments are involved in the Program: the Departments of Cell Biology, Biochemistry, Biology, Genetics, Immunology, Microbiology, Neurobiology, and Pharmacology. Clinical scientists with a strong focus on basic research are members in the program as well. Through the Developmental Biology Training Program, predoctoral students receive classroom and laboratory training that prepares them for competitive research careers. Statistics indicate that most of previous graduates have remained active in research, and suggest that these trainees have been prepared for productive careers in science. The investigators' goal is to build on this tradition of excellence in training.
| Lennox, Ashley L; Mao, Hanqian; Silver, Debra L (2018) RNA on the brain: emerging layers of post-transcriptional regulation in cerebral cortex development. Wiley Interdiscip Rev Dev Biol 7: |
| Waitkus, Matthew S; Pirozzi, Christopher J; Moure, Casey J et al. (2018) Adaptive Evolution of the GDH2 Allosteric Domain Promotes Gliomagenesis by Resolving IDH1R132H-Induced Metabolic Liabilities. Cancer Res 78:36-50 |
| Cordero, Francisco J; Huang, Zhiqing; Grenier, Carole et al. (2017) Histone H3.3K27M Represses p16 to Accelerate Gliomagenesis in a Murine Model of DIPG. Mol Cancer Res 15:1243-1254 |
| Huynh, Nguyen P T; Anderson, Britta A; Guilak, Farshid et al. (2017) Emerging roles for long noncoding RNAs in skeletal biology and disease. Connect Tissue Res 58:116-141 |
| Sayour, Elias J; De Leon, Gabriel; Pham, Christina et al. (2017) Systemic activation of antigen-presenting cells via RNA-loaded nanoparticles. Oncoimmunology 6:e1256527 |
| Linden, Lara M; Gordon, Kacy L; Pani, Ariel M et al. (2017) Identification of regulators of germ stem cell enwrapment by its niche in C. elegans. Dev Biol 429:271-284 |
| Pierpont, Timothy M; Lyndaker, Amy M; Anderson, Claire M et al. (2017) Chemotherapy-Induced Depletion of OCT4-Positive Cancer Stem Cells in a Mouse Model of Malignant Testicular Cancer. Cell Rep 21:1896-1909 |
| Vockley, Christopher M; McDowell, Ian C; D'Ippolito, Antony M et al. (2017) A long-range flexible billboard model of gene activation. Transcription 8:261-267 |
| Miller, Emily E; Kobayashi, Gerson S; Musso, Camila M et al. (2017) EIF4A3 deficient human iPSCs and mouse models demonstrate neural crest defects that underlie Richieri-Costa-Pereira syndrome. Hum Mol Genet 26:2177-2191 |
| Goldman, Joseph Aaron; Kuzu, Guray; Lee, Nutishia et al. (2017) Resolving Heart Regeneration by Replacement Histone Profiling. Dev Cell 40:392-404.e5 |
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