This Interdisciplinary Training Program provides postdoctoral research training in areas relevant to the biological bases of mental disorder. The training program is jointly sponsored by the Department of Psychiatry and the Department of Neuroscience at the Johns Hopkins University School of Medicine. The faculty members all have appointments in either of these two departments and have an established record of collaborative interaction. The research interests of the core faculty represent the major sub-disciplines of the neurosciences and biological psychiatry, including molecular and cellular neurobiology, developmental neurobiology, systems neurobiology, behavioral biology, neuroimaging, psychiatric genetics and neuropsychiatry. The primary vehicle for training is supervised research in a laboratory of one of the core faculty members combined with an organized didactic program in relevant aspects of neuroscience and psychiatric research methodology. The training program provides a unique opportunity for MDs and PhDs to obtain training at the postdoctoral level in multidisciplinary research oriented toward clinical psychiatry. The goal of the program is to prepare future academic researchers to undertake a career in the investigation of the biology of mental disorder.

Agency
National Institute of Health (NIH)
Institute
National Institute of Mental Health (NIMH)
Type
Institutional National Research Service Award (T32)
Project #
5T32MH015330-23
Application #
6185358
Study Section
Special Emphasis Panel (ZMH1-NRB-A (01))
Program Officer
Chavez, Mark
Project Start
1978-07-01
Project End
2004-06-30
Budget Start
2000-07-01
Budget End
2001-06-30
Support Year
23
Fiscal Year
2000
Total Cost
$88,294
Indirect Cost
Name
Johns Hopkins University
Department
Psychiatry
Type
Schools of Medicine
DUNS #
045911138
City
Baltimore
State
MD
Country
United States
Zip Code
21218
Maynard, Kristen R; Hobbs, John W; Phan, BaDoi N et al. (2018) BDNF-TrkB signaling in oxytocin neurons contributes to maternal behavior. Elife 7:
Greenwald, Anthony G; De Houwer, Jan (2017) Unconscious conditioning: Demonstration of existence and difference from conscious conditioning. J Exp Psychol Gen 146:1705-1721
Terrillion, Chantelle E; Francis, T Chase; Puche, Adam C et al. (2017) Decreased Nucleus Accumbens Expression of Psychiatric Disorder Risk Gene Cacna1c Promotes Susceptibility to Social Stress. Int J Neuropsychopharmacol 20:428-433
Wright, C; Shin, J H; Rajpurohit, A et al. (2017) Altered expression of histamine signaling genes in autism spectrum disorder. Transl Psychiatry 7:e1126
Maynard, Kristen R; Hobbs, John W; Sukumar, Mahima et al. (2017) Bdnf mRNA splice variants differentially impact CA1 and CA3 dendrite complexity and spine morphology in the hippocampus. Brain Struct Funct 222:3295-3307
Terrillion, C E; Dao, D T; Cachope, R et al. (2017) Reduced levels of Cacna1c attenuate mesolimbic dopamine system function. Genes Brain Behav 16:495-505
Qian, Xuyu; Nguyen, Ha Nam; Jacob, Fadi et al. (2017) Using brain organoids to understand Zika virus-induced microcephaly. Development 144:952-957
Qian, Xuyu; Nguyen, Ha Nam; Song, Mingxi M et al. (2016) Brain-Region-Specific Organoids Using Mini-bioreactors for Modeling ZIKV Exposure. Cell 165:1238-1254
Xu, Miao; Lee, Emily M; Wen, Zhexing et al. (2016) Identification of small-molecule inhibitors of Zika virus infection and induced neural cell death via a drug repurposing screen. Nat Med 22:1101-1107
Zinkel, S R J; Berkowitz, R I; Stunkard, A J et al. (2016) High energy expenditure is not protective against increased adiposity in children. Pediatr Obes 11:528-534

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