Agency
National Institute of Health (NIH)
Institute
National Institute of Neurological Disorders and Stroke (NINDS)
Type
Institutional National Research Service Award (T32)
Project #
2T32NS007366-06
Application #
6080944
Study Section
NST-2 Subcommittee (NST)
Program Officer
Khachaturian, Henry
Project Start
1995-07-01
Project End
2005-06-30
Budget Start
2000-09-30
Budget End
2001-06-30
Support Year
6
Fiscal Year
2000
Total Cost
$158,143
Indirect Cost
Name
University of Massachusetts Medical School Worcester
Department
Anatomy/Cell Biology
Type
Schools of Medicine
DUNS #
660735098
City
Worcester
State
MA
Country
United States
Zip Code
01655
Roberts-Crowley, Mandy L; Rittenhouse, Ann R (2018) Modulation of CaV1.3b L-type calcium channels by M1 muscarinic receptors varies with CaV? subunit expression. BMC Res Notes 11:681
Dhungel, Nripesh; Eleuteri, Simona; Li, Ling-Bo et al. (2015) Parkinson's disease genes VPS35 and EIF4G1 interact genetically and converge on ?-synuclein. Neuron 85:76-87
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Busza, Ania; Murad, Alejandro; Emery, Patrick (2007) Interactions between circadian neurons control temperature synchronization of Drosophila behavior. J Neurosci 27:10722-33
Gatto, Cheryl L; Walker, Barbara J; Lambert, Stephen (2007) Asymmetric ERM activation at the Schwann cell process tip is required in axon-associated motility. J Cell Physiol 210:122-32
Busza, Ania; Emery-Le, Myai; Rosbash, Michael et al. (2004) Roles of the two Drosophila CRYPTOCHROME structural domains in circadian photoreception. Science 304:1503-6
Gatto, Cheryl L; Walker, Barbara J; Lambert, Stephen (2003) Local ERM activation and dynamic growth cones at Schwann cell tips implicated in efficient formation of nodes of Ranvier. J Cell Biol 162:489-98
Higgins, Cynthia M J; Jung, Cheolwha; Xu, Zuoshang (2003) ALS-associated mutant SOD1G93A causes mitochondrial vacuolation by expansion of the intermembrane space and by involvement of SOD1 aggregation and peroxisomes. BMC Neurosci 4:16
Jung, Cheolwha; Higgins, Cynthia M J; Xu, Zuoshang (2002) Mitochondrial electron transport chain complex dysfunction in a transgenic mouse model for amyotrophic lateral sclerosis. J Neurochem 83:535-45

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