Abstract

The Specific Aims of the Opportunity Fund Management Core (OFMC) of the Stanford CCHI are to: 1) Establish an administrative structure that will facilitate communication with the CCHI Steering Committee regarding priorities and goals for funding; 2) Establish procedures for soliciting, evaluating, prioritizing, and selecting the projects chosen for Opportunity Fund support; 3) Monitor the disbursement and tracking of Opportunity Fund awards, providing reports on the status of the Opportunity Fund at requested intervals or at least annually. 4) Ensure that all projects supported from the Opportunity Fund comply fully with all applicable Federal regulations, policies, and guidelines for research involving human subjects, including the evaluation of risks and protections in projects and appropriate ethical oversight.

Public Health Relevance

The CCHI Opportunity Fund provides a mechanism for support of resources such as technical expertise, access to specific high-cost instrumentation, bioinformatics etc. for cooperative projects involving CCHI investigators. This Opportunity Fund Management Core will follow the recommendations of the CCHI Steering Committee to set the goals, priorities and evaluation criteria for the use of the Opportunity Fund.

  Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of Allergy and Infectious Diseases (NIAID)
Type
Research Program--Cooperative Agreements (U19)
Project #
5U19AI057229-12
Application #
8833770
Study Section
Special Emphasis Panel (ZAI1-LAR-I)
Project Start
Project End
Budget Start
2015-04-01
Budget End
2016-03-31
Support Year
12
Fiscal Year
2015
Total Cost
$1,363,605
Indirect Cost
$518,305

  Institution

Name
Stanford University
Department
Type
DUNS #
009214214
City
Stanford
State
CA
Country
United States
Zip Code
94304

  Publications

Sweeney, Timothy E; Thomas, Neal J; Howrylak, Judie A et al. (2018) Multicohort Analysis of Whole-Blood Gene Expression Data Does Not Form a Robust Diagnostic for Acute Respiratory Distress Syndrome. Crit Care Med 46:244-251
Kronstad, Lisa M; Seiler, Christof; Vergara, Rosemary et al. (2018) Differential Induction of IFN-? and Modulation of CD112 and CD54 Expression Govern the Magnitude of NK Cell IFN-? Response to Influenza A Viruses. J Immunol 201:2117-2131
Wilk, Aaron J; Blish, Catherine A (2018) Diversification of human NK cells: Lessons from deep profiling. J Leukoc Biol 103:629-641
Sweeney, Timothy E; Wynn, James L; Cernada, María et al. (2018) Validation of the Sepsis MetaScore for Diagnosis of Neonatal Sepsis. J Pediatric Infect Dis Soc 7:129-135
Bukhari, Syed Ahmad Chan; O'Connor, Martin J; Martínez-Romero, Marcos et al. (2018) The CAIRR Pipeline for Submitting Standards-Compliant B and T Cell Receptor Repertoire Sequencing Studies to the National Center for Biotechnology Information Repositories. Front Immunol 9:1877
Azad, Tej D; Donato, Michele; Heylen, Line et al. (2018) Inflammatory macrophage-associated 3-gene signature predicts subclinical allograft injury and graft survival. JCI Insight 3:
Leipold, Michael D; Obermoser, Gerlinde; Fenwick, Craig et al. (2018) Comparison of CyTOF assays across sites: Results of a six-center pilot study. J Immunol Methods 453:37-43
Sibener, Leah V; Fernandes, Ricardo A; Kolawole, Elizabeth M et al. (2018) Isolation of a Structural Mechanism for Uncoupling T Cell Receptor Signaling from Peptide-MHC Binding. Cell 174:672-687.e27
Ju, Chia-Hsin; Blum, Lisa K; Kongpachith, Sarah et al. (2018) Plasmablast antibody repertoires in elderly influenza vaccine responders exhibit restricted diversity but increased breadth of binding across influenza strains. Clin Immunol 193:70-79
Sweeney, Timothy E; Perumal, Thanneer M; Henao, Ricardo et al. (2018) A community approach to mortality prediction in sepsis via gene expression analysis. Nat Commun 9:694

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