Rat Liver Tumor Model in Mechanisms of Carginogenesis

Pitot, Henry C.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute of Environmental Health Sciences (NIEHS)
• Type: Research and Development Contracts (N01)
• Project #: N01ES035024-010
• Application #: 3640010
• Project Start: 1983-03-15
• Budget Start: 1988-04-16
• Budget End: 1988-09-30
• Fiscal Year: 1988

Institution

• Name: University of Wisconsin Madison
• Type: Schools of Medicine
• DUNS #: 161202122
• City: Madison
• State: WI
• Country: United States
• Zip Code: 53715

Publications

Hendrich, S; Glauert, H P; Pitot, H C (1986) The phenotypic stability of altered hepatic foci: effects of withdrawal and subsequent readministration of phenobarbital. Carcinogenesis 7:2041-5

Campbell, H A; Xu, Y D; Hanigan, M H et al. (1986) Application of quantitative stereology to the evaluation of phenotypically heterogeneous enzyme-altered foci in the rat liver. J Natl Cancer Inst 76:751-67

Glauert, H P; Beer, D; Rao, M S et al. (1986) Induction of altered hepatic foci in rats by the administration of hypolipidemic peroxisome proliferators alone or following a single dose of diethylnitrosamine. Cancer Res 46:4601-6

Hanigan, M H; Pitot, H C (1985) Growth of carcinogen-altered rat hepatocytes in the liver of syngeneic recipients promoted with phenobarbital. Cancer Res 45:6063-70