Modulation of Dopamine Autoreceptor Function by Cocaine

O'Malley, Karen L.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute on Drug Abuse (NIDA)
• Type: Research Project (R01)
• Project #: 5R01DA008818-02
• Application #: 2121580
• Study Section: Drug Abuse Biomedical Research Review Committee (DABR)
• Project Start: 1995-02-01
• Project End: 1997-12-31
• Budget Start: 1996-02-01
• Budget End: 1996-12-31
• Support Year: 2
• Fiscal Year: 1996

Institution

• Name: Washington University
• Department: Anatomy/Cell Biology
• Type: Schools of Medicine
• DUNS #: 062761671
• City: Saint Louis
• State: MO
• Country: United States
• Zip Code: 63130

Publications

Dalman, F C; O'Malley, K L (1999) kappa-Opioid tolerance and dependence in cultures of dopaminergic midbrain neurons. J Neurosci 19:5750-7

Oh, Y J; Uhland-Smith, A; Kim, J E et al. (1997) Regions outside of the Bcl-2 homology domains, BH1 and BH2 protect a dopaminergic neuronal cell line from staurosporine-induced cell death. Brain Res Mol Brain Res 51:133-42

Yamaguchi, I; Harmon, S K; Todd, R D et al. (1997) The rat D4 dopamine receptor couples to cone transducin (Galphat2) to inhibit forskolin-stimulated cAMP accumulation. J Biol Chem 272:16599-602

O'Hara, C M; Tang, L; Taussig, R et al. (1996) Dopamine D2L receptor couples to G alpha i2 and G alpha i3 but not G alpha i1, leading to the inhibition of adenylate cyclase in transfected cell lines. J Pharmacol Exp Ther 278:354-60