The Administrative/Program Enrichment Core is the administrative arm of the San Antonio Nathan Shock Aging Center. Under the direction of Drs. Arlan Richardson and Randy Strong, the Core will be responsible for the management of the Center, the annual reports, and the evaluation of the Center. In addition, the Administrative/Program Enrichment Core will provide Cores and Center investigators with data management and biostatistical support. This Core also will be the organizational unit responsible for promoting research in basic biology of aging in San Antonio. This will be accomplished through seminars, meetings, and conferences that focus on various aspects of aging. The Administrative/Program Enrichment Core also will be the organizational unit that provides investigators outside the UTHSCSA with access to the services and programs provided by the San Antonio Nathan Shock Aging Center. The goal of our Center will be to administer Research Cores that will become regional/national resources for investigators studying aging. In addition, the Administrative/Program Enrichment Core will be responsible for collaborative efforts with other Nathan Shock Aging Centers.
The Specific Aims of the Administrative/Program Enrichment Core are as follows: 1. To provide administrative/management support for the Center, 2. To conduct evaluations of the Center, 3. To provide data management and biostatistical support to the Research Cores and Center members, 4. To foster an environment that stimulates collaborative efforts in aging at San Antonio, and 5. To promote interactions in aging research and the sharing of Core resources/services with investigators at other institutions and collaboration with other Nathan Shock Centers.
The Administrative/Program Enrichment Core organizational unit of the San Antonio Nathan Shock Center that is responsible for the administration of the Center and promoting research in basic biology of aging in San Antonio. The Administrative/Program Enrichment Core also will be responsible for collaborative efforts with other Nathan Shock Aging Centers
|Branch, Sarah Y; Sharma, Ramaswamy; Beckstead, Michael J (2014) Aging decreases L-type calcium channel currents and pacemaker firing fidelity in substantia nigra dopamine neurons. J Neurosci 34:9310-8|
|Ratnam, Sarayu; Engler, Peter; Bozek, Grazyna et al. (2014) Identification of Ssm1b, a novel modifier of DNA methylation, and its expression during mouse embryogenesis. Development 141:2024-34|
|Zhang, Yiqiang; Bokov, Alex; Gelfond, John et al. (2014) Rapamycin extends life and health in C57BL/6 mice. J Gerontol A Biol Sci Med Sci 69:119-30|
|Edrey, Yael H; Salmon, Adam B (2014) Revisiting an age-old question regarding oxidative stress. Free Radic Biol Med 71:368-78|
|Boiko, Nina; Kucher, Volodymyr; Wang, Bin et al. (2014) Restrictive expression of acid-sensing ion channel 5 (asic5) in unipolar brush cells of the vestibulocerebellum. PLoS One 9:e91326|
|Liu, Yuhong; Diaz, Vivian; Fernandez, Elizabeth et al. (2014) Rapamycin-induced metabolic defects are reversible in both lean and obese mice. Aging (Albany NY) 6:742-54|
|Elbourkadi, Najoua; Austad, Steven N; Miller, Richard A (2014) Fibroblasts from long-lived species of mammals and birds show delayed, but prolonged, phosphorylation of ERK. Aging Cell 13:283-91|
|Hasty, Paul; Livi, Carolina B; Dodds, Sherry G et al. (2014) eRapa restores a normal life span in a FAP mouse model. Cancer Prev Res (Phila) 7:169-78|
|Fok, Wilson C; Bokov, Alex; Gelfond, Jonathan et al. (2014) Combined treatment of rapamycin and dietary restriction has a larger effect on the transcriptome and metabolome of liver. Aging Cell 13:311-9|
|Strong, Randy; Miller, Richard A; Astle, Clinton M et al. (2013) Evaluation of resveratrol, green tea extract, curcumin, oxaloacetic acid, and medium-chain triglyceride oil on life span of genetically heterogeneous mice. J Gerontol A Biol Sci Med Sci 68:6-16|
Showing the most recent 10 out of 144 publications