A diverse and well-trained scientific workforce is necessary to advance biomedical research, improve human health and to develop effective remedies for disease. The University of Missouri-Columbia (MU) is the only four-year comprehensive public university in the State of Missouri and has a critical role in training the next generation of scientists. As part of the University of Missouri-Columbia (MU) commitment to scientific training and diversity, and with prior funding from NIH, we have developed a successful and innovative program to prepare underrepresented minority students for biomedical research. We have three objectives for our Initiative to Maximize Student Diversity (IMSD) program. First, to further develop a supportive and engaged peer community of minority undergraduate students that are interested in exploring biomedical research. We will increase the number of MU freshmen/sophomore students who are exploring a career in biomedical research. Second, we will expand a program targeted at MU junior/senior students that involves these advanced undergraduates in independent research experiences and prepares them for graduate study in the biomedical sciences. Third, we will provide a structured and mentored research environment for minority graduate students to develop into independent scientists as they pursue their PhD degree in a biomedical discipline. Further development of our IMSD program will have a substantial impact on diversity at the University and on our nation's scientific workforce.
A robust biomedical research enterprise is required to improve human health and reduce morbidity and mortality from disease. A diverse scientific workforce, which utilizes the talents and skills of all individuals, is a necessary component of a robust research enterprise. The goal of our program is to increase the diversity of PhD scientists trained in biomedical research.
|Puray-Chavez, Maritza; Tedbury, Philip R; Huber, Andrew D et al. (2017) Multiplex single-cell visualization of nucleic acids and protein during HIV infection. Nat Commun 8:1882|
|Porter, Jay W; Rowles 3rd, Joe L; Fletcher, Justin A et al. (2017) Anti-inflammatory effects of exercise training in adipose tissue do not require FGF21. J Endocrinol 235:97-109|
|Welly, Rebecca J; Liu, Tzu-Wen; Zidon, Terese M et al. (2016) Comparison of Diet versus Exercise on Metabolic Function and Gut Microbiota in Obese Rats. Med Sci Sports Exerc 48:1688-98|
|Figueroa-Cuilan, Wanda; Daniel, Jeremy J; Howell, Matthew et al. (2016) Mini-Tn7 Insertion in an Artificial attTn7 Site Enables Depletion of the Essential Master Regulator CtrA in the Phytopathogen Agrobacterium tumefaciens. Appl Environ Microbiol 82:5015-25|
|Van Gronigen Caesar, Gerialisa; Dale, Jeffrey M; Osman, Erkan Y et al. (2016) Placental development in a mouse model of spinal muscular atrophy. Biochem Biophys Res Commun 470:82-7|
|Mayo, L M; Moore, S G; Poock, S E et al. (2016) Technical note: Validation of a chemical pregnancy test in dairy cows that uses whole blood, shortened incubation times, and visual readout. J Dairy Sci 99:7634-7641|
|Harper, Jennifer L; Caesar, Gerialisa A; Pennington, Kathleen A et al. (2015) Placental changes caused by food restriction during early pregnancy in mice are reversible. Reproduction 150:165-72|
|Zhang, Xinyue; Xu, Xiaojun; Yang, Zhiyu et al. (2015) Mimicking Ribosomal Unfolding of RNA Pseudoknot in a Protein Channel. J Am Chem Soc 137:15742-52|
|Wainright, Katherine S; Fleming, Nicholas J; Rowles, Joe L et al. (2015) Retention of sedentary obese visceral white adipose tissue phenotype with intermittent physical activity despite reduced adiposity. Am J Physiol Regul Integr Comp Physiol 309:R594-602|
|Swatek, Kirby N; Wilson, Rashaun S; Ahsan, Nagib et al. (2014) Multisite phosphorylation of 14-3-3 proteins by calcium-dependent protein kinases. Biochem J 459:15-25|
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