Greene, J G; Sheu, S S; Gross, R A et al. (1998) 3-Nitropropionic acid exacerbates N-methyl-D-aspartate toxicity in striatal culture by multiple mechanisms. Neuroscience 84:503-10
|
Greene, J G; Greenamyre, J T (1996) Manipulation of membrane potential modulates malonate-induced striatal excitotoxicity in vivo. J Neurochem 66:637-43
|
Greene, J G; Porter, R H; Greenamyre, J T (1996) ARL-15896, a novel N-methyl-D-aspartate receptor ion channel antagonist: neuroprotection against mitochondrial metabolic toxicity and regional pharmacology. Exp Neurol 137:66-72
|
Greene, J G; Greenamyre, J T (1995) Exacerbation of NMDA, AMPA, and L-glutamate excitotoxicity by the succinate dehydrogenase inhibitor malonate. J Neurochem 64:2332-8
|
Greene, J G; Greenamyre, J T (1995) Characterization of the excitotoxic potential of the reversible succinate dehydrogenase inhibitor malonate. J Neurochem 64:430-6
|
Greenamyre, J T; Garcia-Osuna, M; Greene, J G (1994) The endogenous cofactors, thioctic acid and dihydrolipoic acid, are neuroprotective against NMDA and malonic acid lesions of striatum. Neurosci Lett 171:17-20
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Porter, R H; Greene, J G; Higgins Jr, D S et al. (1994) Polysynaptic regulation of glutamate receptors and mitochondrial enzyme activities in the basal ganglia of rats with unilateral dopamine depletion. J Neurosci 14:7192-9
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