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Implications of Deficient Gs Coupled Signal Transduction
Schwindinger, William F.
Johns Hopkins University, Baltimore, MD, United States
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Abstract
Funding Agency
Agency
National Institute of Health (NIH)
Institute
National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
Type
Postdoctoral Individual National Research Service Award (F32)
Project #
5F32DK008725-02
Application #
2135586
Study Section
Biological Sciences 2 (BIOL)
Project Start
1993-02-01
Project End
Budget Start
1993-02-01
Budget End
1994-01-31
Support Year
2
Fiscal Year
1993
Total Cost
Indirect Cost
Institution
Name
Johns Hopkins University
Department
Internal Medicine/Medicine
Type
Schools of Medicine
DUNS #
045911138
City
Baltimore
State
MD
Country
United States
Zip Code
21218
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NIH 1993
F32 DK
Implications of Deficient Gs Coupled Signal Transduction
Schwindinger, William F. / Johns Hopkins University
NIH 1992
F32 DK
Implications of Deficient Gs Coupled Signal Transduction
Schwindinger, William F. / Johns Hopkins University
Publications
Schwindinger, W F; Miric, A; Zimmerman, D et al.
(1994)
A novel Gs alpha mutant in a patient with Albright hereditary osteodystrophy uncouples cell surface receptors from adenylyl cyclase.
J Biol Chem 269:25387-91
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