Mechanisms of Hyperpolarization by Endothelial Factors

Murphy, Michael E.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Heart, Lung, and Blood Institute (NHLBI)
• Type: Postdoctoral Individual National Research Service Award (F32)
• Project #: 2F32HL008648-03
• Application #: 2213419
• Study Section: Clinical Trials (CLIN)
• Project Start: 1995-03-26
• Budget Start: 1994-07-01
• Budget End: 1995-06-30
• Support Year: 3
• Fiscal Year: 1994

Institution

• Name: University of Vermont & St Agric College
• Department: Pharmacology
• Type: Schools of Medicine
• DUNS #: 066811191
• City: Burlington
• State: VT
• Country: United States
• Zip Code: 05405

Publications

Jackson, William F (2011) Altered expression and function of ryanodine receptors and FKBP12.6 after subarachnoid hemorrhage: more than meets the eye. J Cereb Blood Flow Metab 31:1-2

Murphy, M E (1999) Ascorbate and dehydroascorbate modulate nitric oxide-induced vasodilations of rat coronary arteries. J Cardiovasc Pharmacol 34:295-303

Murphy, M E; Brayden, J E (1995) Nitric oxide hyperpolarizes rabbit mesenteric arteries via ATP-sensitive potassium channels. J Physiol 486 ( Pt 1):47-58

Murphy, M E; Brayden, J E (1995) Apamin-sensitive K+ channels mediate an endothelium-dependent hyperpolarization in rabbit mesenteric arteries. J Physiol 489 ( Pt 3):723-34