Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
Postdoctoral Individual National Research Service Award (F32)
Project #
1F32HL009444-01
Application #
2214498
Study Section
Experimental Cardiovascular Sciences Study Section (ECS)
Project Start
1996-07-25
Project End
Budget Start
1996-01-29
Budget End
1997-01-28
Support Year
1
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Graduate Hospital (Philadelphia)
Department
Type
DUNS #
City
Philadelphia
State
PA
Country
United States
Zip Code
19146
Zhou, Xueping; Teng, Bunyen; Mustafa, S J (2015) Sex Difference in Coronary Endothelial Dysfunction in Apolipoprotein E Knockout Mouse: Role of NO and A2A Adenosine Receptor. Microcirculation 22:518-27
Zhou, Zhichao; Rajamani, Uthra; Labazi, Hicham et al. (2015) Involvement of NADPH oxidase in A2A adenosine receptor-mediated increase in coronary flow in isolated mouse hearts. Purinergic Signal 11:263-73
Yadav, Vishal R; Nayeem, Mohammed A; Tilley, Stephen L et al. (2015) Angiotensin II stimulation alters vasomotor response to adenosine in mouse mesenteric artery: role for A1 and A2B adenosine receptors. Br J Pharmacol 172:4959-69
Zhou, Xueping; Teng, Bunyen; Tilley, Stephen et al. (2014) Metabolic hyperemia requires ATP-sensitive K+ channels and H2O2 but not adenosine in isolated mouse hearts. Am J Physiol Heart Circ Physiol 307:H1046-55
Zhou, Xueping; Teng, Bunyen; Tilley, Stephen et al. (2013) A1 adenosine receptor negatively modulates coronary reactive hyperemia via counteracting A2A-mediated H2O2 production and KATP opening in isolated mouse hearts. Am J Physiol Heart Circ Physiol 305:H1668-79
Sanjani, Maryam Sharifi; Teng, Bunyen; Krahn, Thomas et al. (2011) Contributions of A2A and A2B adenosine receptors in coronary flow responses in relation to the KATP channel using A2B and A2A/2B double-knockout mice. Am J Physiol Heart Circ Physiol 301:H2322-33