Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
Postdoctoral Individual National Research Service Award (F32)
Project #
1F32HL009781-01
Application #
2027931
Study Section
Experimental Cardiovascular Sciences Study Section (ECS)
Project Start
1997-09-16
Project End
Budget Start
1997-03-17
Budget End
1998-03-16
Support Year
1
Fiscal Year
1997
Total Cost
Indirect Cost
Name
University of Cincinnati
Department
Physiology
Type
Schools of Medicine
DUNS #
City
Cincinnati
State
OH
Country
United States
Zip Code
45221
Sutliff, Roy L; Conforti, Laura; Weber, Craig S et al. (2004) Regulation of the spontaneous contractile activity of the portal vein by the sarcoplasmic reticulum: evidence from the phospholamban gene-ablated mouse. Vascul Pharmacol 41:197-204
Nobe, K; Sutliff, R L; Kranias, E G et al. (2001) Phospholamban regulation of bladder contractility: evidence from gene-altered mouse models. J Physiol 535:867-78
Sutliff, R L; Hoying, J B; Kadambi, V J et al. (1999) Phospholamban is present in endothelial cells and modulates endothelium-dependent relaxation. Evidence from phospholamban gene-ablated mice. Circ Res 84:360-4
Lalli, M J; Shimizu, S; Sutliff, R L et al. (1999) [Ca2+]i homeostasis and cyclic nucleotide relaxation in aorta of phospholamban-deficient mice. Am J Physiol 277:H963-70
Maeda, S; Sutliff, R L; Qian, J et al. (1999) Targeted overexpression of parathyroid hormone-related protein (PTHrP) to vascular smooth muscle in transgenic mice lowers blood pressure and alters vascular contractility. Endocrinology 140:1815-25
Qian, J; Lorenz, J N; Maeda, S et al. (1999) Reduced blood pressure and increased sensitivity of the vasculature to parathyroid hormone-related protein (PTHrP) in transgenic mice overexpressing the PTH/PTHrP receptor in vascular smooth muscle. Endocrinology 140:1826-33
Sutliff, R L; Weber, C S; Qian, J et al. (1999) Vasorelaxant properties of parathyroid hormone-related protein in the mouse: evidence for endothelium involvement independent of nitric oxide formation. Endocrinology 140:2077-83