Growth Factors in Human Fetal Oogenesis

Yeh, John

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: Eunice Kennedy Shriver National Institute of Child Health & Human Development (NICHD)
• Type: Clinical Investigator Award (CIA) (K08)
• Project #: 7K08HD001016-06
• Application #: 2670531
• Study Section: Population Research Committee (HDPR)
• Program Officer: Parrott, Estella C
• Project Start: 1993-07-01
• Project End: 2000-03-31
• Budget Start: 1999-09-01
• Budget End: 2000-03-31
• Support Year: 6
• Fiscal Year: 1999

Institution

• Name: University of Minnesota Twin Cities
• Department: Obstetrics & Gynecology
• Type: Schools of Medicine
• DUNS #: 168559177
• City: Minneapolis
• State: MN
• Country: United States
• Zip Code: 55455

Publications

Khan, Shah M; Oliver, Rush H; Yeh, John (2005) Epidermal growth factor receptor inhibition by tyrphostin 51 induces apoptosis in luteinized granulosa cells. J Clin Endocrinol Metab 90:469-73

Khan, S M; Oliver, R H; Dauffenbach, L M et al. (2000) Depletion of Raf-1 protooncogene by geldanamycin causes apoptosis in human luteinized granulosa cells. Fertil Steril 74:359-65

Schilling, B; Yeh, J (1999) Expression of transforming growth factor (TGF)-beta1, TGF-beta2, and TGF-beta3 and of type I and II TGF-beta receptors during the development of the human fetal ovary. Fertil Steril 72:147-53

Bennett, R A; Osathanondh, R; Yeh, J (1996) Immunohistochemical localization of transforming growth factor-alpha, epidermal growth factor (EGF), and EGF receptor in the human fetal ovary. J Clin Endocrinol Metab 81:3073-6