Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
Clinical Investigator Award (CIA) (K08)
Project #
5K08HL002972-04
Application #
2210867
Study Section
Research Training Review Committee (RTR)
Project Start
1994-02-01
Project End
1999-01-31
Budget Start
1996-02-01
Budget End
1997-01-31
Support Year
4
Fiscal Year
1996
Total Cost
Indirect Cost

  Institution

Name
University of Michigan Ann Arbor
Department
Genetics
Type
Schools of Medicine
DUNS #
791277940
City
Ann Arbor
State
MI
Country
United States
Zip Code
48109

  Publications

Chen, Kejian; Godfrey, Donald A; Ilyas, Omer et al. (2009) Cerebellum-related characteristics of Scn8a-mutant mice. Cerebellum 8:192-201
Chen, K; Sprunger, L K; Meisler, M H et al. (1999) Reduced spontaneous activity in the dorsal cochlear nucleus of Scn8a mutant mice. Brain Res 847:85-9
Sprunger, L K; Escayg, A; Tallaksen-Greene, S et al. (1999) Dystonia associated with mutation of the neuronal sodium channel Scn8a and identification of the modifier locus Scnm1 on mouse chromosome 3. Hum Mol Genet 8:471-9
Plummer, N W; Galt, J; Jones, J M et al. (1998) Exon organization, coding sequence, physical mapping, and polymorphic intragenic markers for the human neuronal sodium channel gene SCN8A. Genomics 54:287-96
Garcia, K D; Sprunger, L K; Meisler, M H et al. (1998) The sodium channel Scn8a is the major contributor to the postnatal developmental increase of sodium current density in spinal motoneurons. J Neurosci 18:5234-9
Harm, D L; Kotses, H; Creer, T L (1985) Improving the ability of peak expiratory flow rates to predict asthma. J Allergy Clin Immunol 76:688-94