Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
Clinical Investigator Award (CIA) (K08)
Project #
1K08HL003475-01
Application #
2211742
Study Section
Research Training Review Committee (RTR)
Project Start
1996-08-01
Project End
2001-07-31
Budget Start
1996-08-01
Budget End
1997-07-31
Support Year
1
Fiscal Year
1996
Total Cost
Indirect Cost

  Institution

Name
Northern California Institute Research & Education
Department
Type
DUNS #
City
San Francisco
State
CA
Country
United States
Zip Code
94121

  Publications

Greyson, Clifford R; Schwartz, Gregory G; Lu, Li et al. (2008) Calpain inhibition attenuates right ventricular contractile dysfunction after acute pressure overload. J Mol Cell Cardiol 44:59-68
Lu, L; Xu, Y; Zhu, P et al. (2001) A common mechanism for concurrent changes of diastolic muscle length and systolic function in intact hearts. Am J Physiol Heart Circ Physiol 280:H1513-8
Zhu, P; Lu, L; Xu, Y et al. (2000) Glucose-insulin-potassium preserves systolic and diastolic function in ischemia and reperfusion in pigs. Am J Physiol Heart Circ Physiol 278:H595-603
Greyson, C; Xu, Y; Lu, L et al. (2000) Right ventricular pressure and dilation during pressure overload determine dysfunction after pressure overload. Am J Physiol Heart Circ Physiol 278:H1414-20
Lu, L; Xu, Y; Greyson, C R et al. (1999) Non-elastic deformation of myocardium in low-flow ischemia and reperfusion: ultrastructure-function relations. J Mol Cell Cardiol 31:1157-69
Greyson, C; Xu, Y; Cohen, J et al. (1997) Right ventricular dysfunction persists following brief right ventricular pressure overload. Cardiovasc Res 34:281-8