Agency
National Institute of Health (NIH)
Institute
National Institute of Allergy and Infectious Diseases (NIAID)
Type
Career Transition Award (K22)
Project #
5K22AI053836-02
Application #
6640759
Study Section
Microbiology and Infectious Diseases B Subcommittee (MID)
Program Officer
Beisel, Christopher E
Project Start
2002-05-01
Project End
2005-03-31
Budget Start
2003-05-01
Budget End
2005-03-31
Support Year
2
Fiscal Year
2003
Total Cost
$107,488
Indirect Cost
Name
University of Illinois at Chicago
Department
Ophthalmology
Type
Schools of Medicine
DUNS #
098987217
City
Chicago
State
IL
Country
United States
Zip Code
60612
Tiwari, Vaibhav; Shukla, Deepak (2010) Phosphoinositide 3 kinase signalling may affect multiple steps during herpes simplex virus type-1 entry. J Gen Virol 91:3002-9
Clement, Christian; Tiwari, Vaibhav; Scanlan, Perry M et al. (2006) A novel role for phagocytosis-like uptake in herpes simplex virus entry. J Cell Biol 174:1009-21
Scanlan, Perry M; Tiwari, Vaibhav; Bommireddy, Susmita et al. (2005) Spinoculation of heparan sulfate deficient cells enhances HSV-1 entry, but does not abolish the need for essential glycoproteins in viral fusion. J Virol Methods 128:104-12
Xu, Ding; Tiwari, Vaibhav; Xia, Guoqing et al. (2005) Characterization of heparan sulphate 3-O-sulphotransferase isoform 6 and its role in assisting the entry of herpes simplex virus type 1. Biochem J 385:451-9
Tiwari, Vaibhav; Clement, Christian; Duncan, Michael B et al. (2004) A role for 3-O-sulfated heparan sulfate in cell fusion induced by herpes simplex virus type 1. J Gen Virol 85:805-9
Valyi-Nagy, Tibor; Sheth, Veeral; Clement, Christian et al. (2004) Herpes simplex virus entry receptor nectin-1 is widely expressed in the murine eye. Curr Eye Res 29:303-9
Scanlan, Perry M; Tiwari, Vaibhav; Bommireddy, Susmita et al. (2003) Cellular expression of gH confers resistance to herpes simplex virus type-1 entry. Virology 312:14-24
Xia, Guoqing; Chen, Jinghua; Tiwari, Vaibhav et al. (2002) Heparan sulfate 3-O-sulfotransferase isoform 5 generates both an antithrombin-binding site and an entry receptor for herpes simplex virus, type 1. J Biol Chem 277:37912-9