Agency
National Institute of Health (NIH)
Institute
National Institute on Alcohol Abuse and Alcoholism (NIAAA)
Type
Research Project (R01)
Project #
5R01AA010058-02
Application #
2046502
Study Section
Biochemistry, Physiology and Medicine Subcommittee (ALCB)
Project Start
1995-02-01
Project End
1998-01-31
Budget Start
1996-03-01
Budget End
1997-01-31
Support Year
2
Fiscal Year
1996
Total Cost
Indirect Cost
Name
Northwestern University at Chicago
Department
Microbiology/Immun/Virology
Type
Schools of Dentistry
DUNS #
005436803
City
Chicago
State
IL
Country
United States
Zip Code
60611
Latif, Omar; Peterson, Jeffrey D; Waltenbaugh, Carl (2002) Alcohol-mediated polarization of type 1 and type 2 immune responses. Front Biosci 7:a135-47
Ostrovidov, Serge; Howard, Laurence M; Ikeda, Masato et al. (2002) Restoration of ethanol-compromised T(h)1 responses by sodium orthovanadate. Int Immunol 14:1239-45
Waltenbaugh, C; Vasquez, K; Peterson, J D (1998) Alcohol consumption alters antigen-specific Th1 responses: mechanisms of deficit and repair. Alcohol Clin Exp Res 22:220S-223S
Peterson, J D; Vasquez, K; Waltenbaugh, C (1998) Interleukin-12 therapy restores cell-mediated immunity in ethanol-consuming mice. Alcohol Clin Exp Res 22:245-51
Peterson, J D; Herzenberg, L A; Vasquez, K et al. (1998) Glutathione levels in antigen-presenting cells modulate Th1 versus Th2 response patterns. Proc Natl Acad Sci U S A 95:3071-6
Waltenbaugh, C; Peterson, J D (1997) Ethanol impairs the induction of delayed hypersensitivity in C57BL/6 mice. Alcohol 14:149-53
Schodde, H; Hurst, S; Munroe, M et al. (1996) Ethanol ingestion inhibits cell-mediated immune responses of unprimed T-cell receptor transgenic mice. Alcohol Clin Exp Res 20:890-9