Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute on Alcohol Abuse and Alcoholism (NIAAA)
Type
Research Project (R01)
Project #
5R01AA010121-02
Application #
2046610
Study Section
Clinical and Treatment Subcommittee (ALCP)
Project Start
1995-01-01
Project End
1997-12-31
Budget Start
1996-01-01
Budget End
1996-12-31
Support Year
2
Fiscal Year
1996
Total Cost
Indirect Cost

  Institution

Name
Yale University
Department
Psychiatry
Type
Schools of Medicine
DUNS #
082359691
City
New Haven
State
CT
Country
United States
Zip Code
06520

  Publications

Petrakis, Ismene L; Limoncelli, Diana; Gueorguieva, Ralitza et al. (2004) Altered NMDA glutamate receptor antagonist response in individuals with a family vulnerability to alcoholism. Am J Psychiatry 161:1776-82
Krystal, John H; Petrakis, Ismene L; Limoncelli, Diana et al. (2003) Altered NMDA glutamate receptor antagonist response in recovering ethanol-dependent patients. Neuropsychopharmacology 28:2020-8
Steinmetz, J E; Blankenship, M R; Green, J T et al. (2000) Evaluation of behavioral disinhibition in P/NP and HAD1/LAD1 rats. Prog Neuropsychopharmacol Biol Psychiatry 24:1025-39
Behar, K L; Rothman, D L; Petersen, K F et al. (1999) Preliminary evidence of low cortical GABA levels in localized 1H-MR spectra of alcohol-dependent and hepatic encephalopathy patients. Am J Psychiatry 156:952-4
Krystal, J H; Petrakis, I L; Webb, E et al. (1998) Dose-related ethanol-like effects of the NMDA antagonist, ketamine, in recently detoxified alcoholics. Arch Gen Psychiatry 55:354-60
Manor, D; Rothman, D L; Mason, G F et al. (1996) The rate of turnover of cortical GABA from [1-13C]glucose is reduced in rats treated with the GABA-transaminase inhibitor vigabatrin (gamma-vinyl GABA). Neurochem Res 21:1031-41