Novel Hormone Delivery System for Treating Osteopenia

Wronski, Thomas J.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute on Aging (NIA)
• Type: Research Project (R01)
• Project #: 5R01AG011441-04
• Application #: 2052631
• Study Section: Orthopedics and Musculoskeletal Study Section (ORTH)
• Project Start: 1993-09-30
• Project End: 1998-07-31
• Budget Start: 1996-08-01
• Budget End: 1997-07-31
• Support Year: 4
• Fiscal Year: 1996

Institution

• Name: University of Florida
• Department: Physiology
• Type: Schools of Veterinary Medicine
• DUNS #: 073130411
• City: Gainesville
• State: FL
• Country: United States
• Zip Code: 32611

Publications

Li, M; Liang, H; Shen, Y et al. (1999) Parathyroid hormone stimulates cancellous bone formation at skeletal sites regardless of marrow composition in ovariectomized rats. Bone 24:95-100

Wronski, T J; Pun, S; Liang, H (1999) Effects of age, estrogen depletion, and parathyroid hormone treatment on vertebral cancellous wall width in female rats. Bone 25:465-8

Shen, Y; Li, M; Wronski, T J (1997) Calcitonin provides complete protection against cancellous bone loss in the femoral neck of ovariectomized rats. Calcif Tissue Int 60:457-61

Shen, Y; Li, M; Wronski, T J (1996) Skeletal effects of calcitonin treatment and withdrawal in ovariectomized rats. Calcif Tissue Int 58:263-7

Li, M; Shen, Y; Burton, K W et al. (1996) A comparison of the skeletal effects of intermittent and continuous administration of calcitonin in ovariectomized rats. Bone 18:375-80