HIV in Infants-Early Immunity and Genetic Susceptibility

Cloyd, Miles W.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute of Allergy and Infectious Diseases (NIAID)
• Type: Research Project (R01)
• Project #: 3R01AI032444-02S1
• Application #: 3147530
• Study Section: AIDS and Related Research Study Section 1 (ARRA)
• Project Start: 1991-09-30
• Project End: 1995-07-31
• Budget Start: 1992-09-01
• Budget End: 1993-07-31
• Support Year: 2
• Fiscal Year: 1992

Institution

• Name: University of Texas Medical Br Galveston
• Type: Schools of Medicine
• DUNS #: 041367053
• City: Galveston
• State: TX
• Country: United States
• Zip Code: 77555

Publications

Wang, L; Klimpel, G R; Planas, J M et al. (1998) Apoptotic killing of CD4+ T lymphocytes in HIV-1-infected PHA-stimulated PBL cultures is mediated by CD8+ LAK cells. Virology 241:169-80

Song, S K; Li, H; Cloyd, M W (1996) Rates of shutdown of HIV-1 into latency: roles of the LTR and tat/rev/vpu gene region. Virology 225:377-86

Li, X D; Moore, B; Cloyd, M W (1996) Gradual shutdown of virus production resulting in latency is the norm during the chronic phase of human immunodeficiency virus replication and differential rates and mechanisms of shutdown are determined by viral sequences. Virology 225:196-212

Moore, B E (1993) Survival of human immunodeficiency virus (HIV), HIV-infected lymphocytes, and poliovirus in water. Appl Environ Microbiol 59:1437-43