Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of Allergy and Infectious Diseases (NIAID)
Type
Research Project (R01)
Project #
5R01AI033444-03
Application #
2068453
Study Section
Bacteriology and Mycology Subcommittee 2 (BM)
Project Start
1994-05-01
Project End
1997-04-30
Budget Start
1996-05-01
Budget End
1997-04-30
Support Year
3
Fiscal Year
1996
Total Cost
Indirect Cost

  Institution

Name
Harvard University
Department
Microbiology/Immun/Virology
Type
Schools of Medicine
DUNS #
082359691
City
Boston
State
MA
Country
United States
Zip Code
02115

  Publications

Schechter, Lisa M; Jain, Sumita; Akbar, Samina et al. (2003) The small nucleoid-binding proteins H-NS, HU, and Fis affect hilA expression in Salmonella enterica serovar Typhimurium. Infect Immun 71:5432-5
Rodriguez, Christine R; Schechter, Lisa M; Lee, Catherine A (2002) Detection and characterization of the S. typhimurium HilA protein. BMC Microbiol 2:31
Lostroh, C P; Lee, C A (2001) The Salmonella pathogenicity island-1 type III secretion system. Microbes Infect 3:1281-91
Lostroh, C P; Lee, C A (2001) The HilA box and sequences outside it determine the magnitude of HilA-dependent activation of P(prgH) from Salmonella pathogenicity island 1. J Bacteriol 183:4876-85
Lucas, R L; Lee, C A (2001) Roles of hilC and hilD in regulation of hilA expression in Salmonella enterica serovar Typhimurium. J Bacteriol 183:2733-45
Lucas, R L; Lee, C A (2000) Unravelling the mysteries of virulence gene regulation in Salmonella typhimurium. Mol Microbiol 36:1024-33
Lostroh, C P; Bajaj, V; Lee, C A (2000) The cis requirements for transcriptional activation by HilA, a virulence determinant encoded on SPI-1. Mol Microbiol 37:300-15
Murray, R A; Lee, C A (2000) Invasion genes are not required for Salmonella enterica serovar typhimurium to breach the intestinal epithelium: evidence that salmonella pathogenicity island 1 has alternative functions during infection. Infect Immun 68:5050-5
Schechter, L M; Damrauer, S M; Lee, C A (1999) Two AraC/XylS family members can independently counteract the effect of repressing sequences upstream of the hilA promoter. Mol Microbiol 32:629-42
Lee, C A (1997) Type III secretion systems: machines to deliver bacterial proteins into eukaryotic cells? Trends Microbiol 5:148-56

Showing the most recent 10 out of 14 publications