Alpha Beta T Cell Independent B Cell Function

Tigelaar, Robert E.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute of Allergy and Infectious Diseases (NIAID)
• Type: Research Project (R01)
• Project #: 1R01AI038932-01A1
• Application #: 2076048
• Study Section: Immunological Sciences Study Section (IMS)
• Project Start: 1996-07-15
• Project End: 2001-06-30
• Budget Start: 1996-07-15
• Budget End: 1997-06-30
• Support Year: 1
• Fiscal Year: 1996

Institution

• Name: Yale University
• Department: Biology
• Type: Schools of Arts and Sciences
• DUNS #: 082359691
• City: New Haven
• State: CT
• Country: United States
• Zip Code: 06520

Publications

Smith, A L; Hayday, A C (2000) Genetic dissection of primary and secondary responses to a widespread natural pathogen of the gut, Eimeria vermiformis. Infect Immun 68:6273-80

Wen, L; Barber, D F; Pao, W et al. (1998) Primary gamma delta cell clones can be defined phenotypically and functionally as Th1/Th2 cells and illustrate the association of CD4 with Th2 differentiation. J Immunol 160:1965-74

Dianda, L; Hanby, A M; Wright, N A et al. (1997) T cell receptor-alpha beta-deficient mice fail to develop colitis in the absence of a microbial environment. Am J Pathol 150:91-7

Pao, W; Wen, L; Smith, A L et al. (1996) Gamma delta T cell help of B cells is induced by repeated parasitic infection, in the absence of other T cells. Curr Biol 6:1317-25