Agency
National Institute of Health (NIH)
Institute
National Institute of Allergy and Infectious Diseases (NIAID)
Type
Research Project (R01)
Project #
1R01AI040003-01
Application #
2076947
Study Section
AIDS and Related Research Study Section 3 (ARRC)
Project Start
1996-09-01
Project End
2000-08-31
Budget Start
1996-09-01
Budget End
1997-08-31
Support Year
1
Fiscal Year
1996
Total Cost
Indirect Cost
Name
National Jewish Health
Department
Type
DUNS #
City
Denver
State
CO
Country
United States
Zip Code
80206
Wang, Jiangfang; Reuschel, Emma L; Shackelford, Jason M et al. (2011) HIV-1 Vif promotes the Gýýý- to S-phase cell-cycle transition. Blood 117:1260-9
Wang, Jiangfang; Shackelford, Jason M; Selliah, Nithianandan et al. (2008) The HIV-1 Vif protein mediates degradation of Vpr and reduces Vpr-induced cell cycle arrest. DNA Cell Biol 27:267-77
Wang, Jiangfang; Shackelford, Jason M; Casella, Carolyn R et al. (2007) The Vif accessory protein alters the cell cycle of human immunodeficiency virus type 1 infected cells. Virology 359:243-52
Yin, Jiyi; Ma, Zhengyu; Selliah, Nithianandan et al. (2006) Effective gene suppression using small interfering RNA in hard-to-transfect human T cells. J Immunol Methods 312:1-11
Yin, Jiyi; Chen, Maria F; Finkel, Terri H (2004) Differential gene expression during HIV-1 infection analyzed by suppression subtractive hybridization. AIDS 18:587-96
Wang, JiangFang; Marschner, Susanne; Finkel, Terri H (2004) CXCR4 engagement is required for HIV-1-induced L-selectin shedding. Blood 103:1218-21
Selliah, Nithianandan; Shackelford, Jason; Wang, Jiang-Fang et al. (2003) T cell signaling and apoptosis in HIV disease. Immunol Res 27:247-60
Marschner, Susanne; Hunig, Thomas; Cambier, John C et al. (2002) Ligation of human CD4 interferes with antigen-induced activation of primary T cells. Immunol Lett 82:131-9
Selliah, N; Finkel, T H (2001) HIV-1 NL4-3, but not IIIB, inhibits JAK3/STAT5 activation in CD4(+) T cells. Virology 286:412-21
Marschner, S; Freiberg, B A; Kupfer, A et al. (1999) Ligation of the CD4 receptor induces activation-independent down-regulation of L-selectin. Proc Natl Acad Sci U S A 96:9763-8

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