Expression of Antimycobacterial Immunity

North, Robert John

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute of Allergy and Infectious Diseases (NIAID)
• Type: Research Project (R01)
• Project #: 1R01AI040071-01
• Application #: 2077006
• Study Section: Bacteriology and Mycology Subcommittee 2 (BM)
• Project Start: 1996-09-01
• Project End: 1999-08-31
• Budget Start: 1996-09-01
• Budget End: 1997-08-31
• Support Year: 1
• Fiscal Year: 1996

Institution

• Name: Trudeau Institute, Inc.
• City: Saranac Lake
• State: NY
• Country: United States
• Zip Code: 12983

Publications

North, R J; LaCourse, R; Ryan, L et al. (1999) Consequence of Nramp1 deletion to Mycobacterium tuberculosis infection in mice. Infect Immun 67:5811-4

Medina, E; North, R J (1999) Genetically susceptible mice remain proportionally more susceptible to tuberculosis after vaccination. Immunology 96:16-21

North, R J; Ryan, L; LaCource, R et al. (1999) Growth rate of mycobacteria in mice as an unreliable indicator of mycobacterial virulence. Infect Immun 67:5483-5

North, R J; LaCourse, R; Ryan, L (1999) Vaccinated mice remain more susceptible to Mycobacterium tuberculosis infection initiated via the respiratory route than via the intravenous route. Infect Immun 67:2010-2

North, R J; Medina, E (1998) How important is Nramp1 in tuberculosis? Trends Microbiol 6:441-3

North, R J (1998) Mice incapable of making IL-4 or IL-10 display normal resistance to infection with Mycobacterium tuberculosis. Clin Exp Immunol 113:55-8

Medina, E; North, R J (1998) Resistance ranking of some common inbred mouse strains to Mycobacterium tuberculosis and relationship to major histocompatibility complex haplotype and Nramp1 genotype. Immunology 93:270-4

North, R J; Medina, E (1996) Significance of the antimicrobial resistance gene, Nramp1, in resistance to virulent Mycobacterium tuberculosis infection. Res Immunol 147:493-9