Agency
National Institute of Health (NIH)
Institute
National Institute of Allergy and Infectious Diseases (NIAID)
Type
Research Project (R01)
Project #
7R01AI040847-02
Application #
2672924
Study Section
AIDS and Related Research Study Section 1 (ARRA)
Project Start
1997-09-01
Project End
2002-08-31
Budget Start
1997-12-01
Budget End
1998-11-30
Support Year
2
Fiscal Year
1997
Total Cost
Indirect Cost
Name
J. David Gladstone Institutes
Department
Type
DUNS #
047120084
City
San Francisco
State
CA
Country
United States
Zip Code
94158
Kaehlcke, Katrin; Dorr, Alexander; Hetzer-Egger, Claudia et al. (2003) Acetylation of Tat defines a cyclinT1-independent step in HIV transactivation. Mol Cell 12:167-76
Dorr, Alexander; Kiermer, Veronique; Pedal, Angelika et al. (2002) Transcriptional synergy between Tat and PCAF is dependent on the binding of acetylated Tat to the PCAF bromodomain. EMBO J 21:2715-23
Ott, M; Lovett, J L; Mueller, L et al. (1998) Superinduction of IL-8 in T cells by HIV-1 Tat protein is mediated through NF-kappaB factors. J Immunol 160:2872-80
Herbein, G; Van Lint, C; Lovett, J L et al. (1998) Distinct mechanisms trigger apoptosis in human immunodeficiency virus type 1-infected and in uninfected bystander T lymphocytes. J Virol 72:660-70
Emiliani, S; Fischle, W; Ott, M et al. (1998) Mutations in the tat gene are responsible for human immunodeficiency virus type 1 postintegration latency in the U1 cell line. J Virol 72:1666-70