Neuromuscluar Immunopathology in Eamg-Resistant Rats

Krolick, Keith A.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS)
• Type: Research Project (R01)
• Project #: 5R01AR041441-04
• Application #: 2080698
• Study Section: Neurology C Study Section (NEUC)
• Project Start: 1993-05-01
• Project End: 1998-04-30
• Budget Start: 1996-05-01
• Budget End: 1998-04-30
• Support Year: 4
• Fiscal Year: 1996

Institution

• Name: University of Texas Health Science Center San Antonio
• Department: Microbiology/Immun/Virology
• Type: Schools of Medicine
• DUNS #: 800772162
• City: San Antonio
• State: TX
• Country: United States
• Zip Code: 78229

Publications

Krolick, K A; Yeh, T M; Edlund, S A (1996) Lewis rats given antibodies against denatured acetylcholine receptor become resistant to induction of experimental autoimmune myasthenia gravis. Cell Immunol 172:10-20

Zoda, T E; Brandon, K; Krolick, K A (1995) Neonatal tolerance to an immunodominant T cell reactivity does not confer resistance to EAMG induction in Lewis rats. J Neuroimmunol 57:35-44

Thompson, P A; Edlund, S A; Krolick, K A (1995) Identification of an idiotype associated with antibodies responsible for neuromuscular dysfunction in rats with experimental autoimmune myasthenia gravis. Clin Immunol Immunopathol 75:57-67