Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of Arthritis and Musculoskeletal and Skin Diseases (NIAMS)
Type
Research Project (R01)
Project #
1R01AR043773-01
Application #
2006527
Study Section
Immunological Sciences Study Section (IMS)
Project Start
1996-09-30
Project End
2001-08-31
Budget Start
1996-09-30
Budget End
1997-08-31
Support Year
1
Fiscal Year
1996
Total Cost
Indirect Cost

  Institution

Name
Tufts University
Department
Pathology
Type
Schools of Medicine
DUNS #
604483045
City
Boston
State
MA
Country
United States
Zip Code
02111

  Publications

Berland, Robert; Fiering, Steven; Wortis, Henry H (2010) A conserved enhancer element differentially regulates developmental expression of CD5 in B and T cells. J Immunol 185:7537-43
Jin, Lei; McLean, Paul A; Neel, Benjamin G et al. (2002) Sialic acid binding domains of CD22 are required for negative regulation of B cell receptor signaling. J Exp Med 195:1199-205
Ma, Limei; Wortis, Henry H; Kenter, Amy L (2002) Two new isotype-specific switching activities detected for Ig class switching. J Immunol 168:2835-46
Lin, S C; Wortis, H H; Stavnezer, J (1998) The ability of CD40L, but not lipopolysaccharide, to initiate immunoglobulin switching to immunoglobulin G1 is explained by differential induction of NF-kappaB/Rel proteins. Mol Cell Biol 18:5523-32
Nadler, M J; McLean, P A; Neel, B G et al. (1997) B cell antigen receptor-evoked calcium influx is enhanced in CD22-deficient B cell lines. J Immunol 159:4233-43
Nadler, M J; Chen, B; Anderson, J S et al. (1997) Protein-tyrosine phosphatase SHP-1 is dispensable for FcgammaRIIB-mediated inhibition of B cell antigen receptor activation. J Biol Chem 272:20038-43