Agency
National Institute of Health (NIH)
Institute
National Cancer Institute (NCI)
Type
Research Project (R01)
Project #
3R01CA014609-12S1
Application #
3163971
Study Section
Cellular Biology and Physiology Subcommittee 1 (CBY)
Project Start
1979-06-01
Project End
1987-11-30
Budget Start
1984-12-01
Budget End
1985-11-30
Support Year
12
Fiscal Year
1985
Total Cost
Indirect Cost
Name
University of Texas Sw Medical Center Dallas
Department
Type
Overall Medical
DUNS #
City
Dallas
State
TX
Country
United States
Zip Code
75390
Altankov, G; Grinnell, F (1993) Depletion of intracellular potassium disrupts coated pits and reversibly inhibits cell polarization during fibroblast spreading. J Cell Biol 120:1449-59
Grinnell, F; Ho, C H; Wysocki, A (1992) Degradation of fibronectin and vitronectin in chronic wound fluid: analysis by cell blotting, immunoblotting, and cell adhesion assays. J Invest Dermatol 98:410-6
Grinnell, F (1992) Wound repair, keratinocyte activation and integrin modulation. J Cell Sci 101 ( Pt 1):1-5
Mochitate, K; Pawelek, P; Grinnell, F (1991) Stress relaxation of contracted collagen gels: disruption of actin filament bundles, release of cell surface fibronectin, and down-regulation of DNA and protein synthesis. Exp Cell Res 193:198-207
Guo, M; Kim, L T; Akiyama, S K et al. (1991) Altered processing of integrin receptors during keratinocyte activation. Exp Cell Res 195:315-22
Grinnell, F; Backman, R (1991) Role of integrin receptors in manganese-dependent BHK cell spreading on albumin-coated substrata. Exp Cell Res 195:218-23
Guo, M; Toda, K; Grinnell, F (1990) Activation of human keratinocyte migration on type I collagen and fibronectin. J Cell Sci 96 ( Pt 2):197-205
Grinnell, F (1990) The activated keratinocyte: up regulation of cell adhesion and migration during wound healing. J Trauma 30:S144-9
Tuan, T L; Grinnell, F (1989) Fibronectin and fibrinolysis are not required for fibrin gel contraction by human skin fibroblasts. J Cell Physiol 140:577-83
Grinnell, F; Ho, C H; Tuan, T L (1988) Cell adhesion and phagocytosis promoted by monoclonal antibodies not directed against fibronectin receptors. J Cell Sci 90 ( Pt 2):201-14

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