Hormone Production by Pituitary Tumor Cells

Biswas, Debajit K.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Cancer Institute (NCI)
• Type: Research Project (R01)
• Project #: 3R01CA028218-03S4
• Application #: 3168033
• Study Section: Endocrinology Study Section (END)
• Project Start: 1980-12-01
• Project End: 1984-12-31
• Budget Start: 1982-12-01
• Budget End: 1984-12-31
• Support Year: 3
• Fiscal Year: 1985

Institution

• Name: Harvard University
• Type: Schools of Dentistry/Oral Hygn
• DUNS #: 082359691
• City: Boston
• State: MA
• Country: United States

Publications

Zhang, Z X; Kumar, V; Rivera, R T et al. (1990) cis-acting negative regulatory element of prolactin gene. J Biol Chem 265:4785-8

Zhang, Z X; Kumar, V; Rivera, R T et al. (1989) Suppression of prolactin gene expression in GH cells correlates with site-specific DNA methylation. DNA 8:605-13

Kumar, V; Biswas, D K (1988) Dynamic state of site-specific DNA methylation concurrent to altered prolactin and growth hormone gene expression in the pituitary gland of pregnant and lactating rats. J Biol Chem 263:12645-52

Pasion, S G; Hartigan, J A; Kumar, V et al. (1987) DNA sequence responsible for the amplification of adjacent genes. DNA 6:419-28

Kumar, V; Wong, D T; Pasion, S G et al. (1987) Defective distal regulatory element at the 5' upstream of rat prolactin gene of steroid-nonresponsive GH-subclone. Biochim Biophys Acta 910:213-23