Mechanisms of Regulation of Cell Proliferation

Getz, Michael J.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Cancer Institute (NCI)
• Type: Research Project (R01)
• Project #: 3R01CA033643-03S1
• Application #: 3171442
• Study Section: Molecular Biology Study Section (MBY)
• Project Start: 1983-02-01
• Project End: 1986-02-28
• Budget Start: 1985-02-01
• Budget End: 1986-02-28
• Support Year: 3
• Fiscal Year: 1986

Institution

• Name: Mayo Clinic, Rochester
• City: Rochester
• State: MN
• Country: United States
• Zip Code: 55905

Publications

Eldredge, E R; Korf, G M; Christensen, T A et al. (1994) Activation of c-fos gene expression by a kinase-deficient epidermal growth factor receptor. Mol Cell Biol 14:7527-34

Foster, D N; Min, B; Foster, L K et al. (1992) Positive and negative cis-acting regulatory elements mediate expression of the mouse vascular smooth muscle alpha-actin gene. J Biol Chem 267:11995-2003

Stoflet, E S; Schmidt, L J; Elder, P K et al. (1992) Activation of a muscle-specific actin gene promoter in serum-stimulated fibroblasts. Mol Biol Cell 3:1073-83

Ranganathan, G; Getz, M J (1990) Cooperative stimulation of specific gene transcription by epidermal growth factor and transforming growth factor type beta 1. J Biol Chem 265:3001-4

Elder, P K; French, C L; Subramaniam, M et al. (1988) Evidence that the functional beta-actin gene is single copy in most mice and is associated with 5' sequences capable of conferring serum- and cycloheximide-dependent regulation. Mol Cell Biol 8:480-5

Blatti, S P; Foster, D N; Ranganathan, G et al. (1988) Induction of fibronectin gene transcription and mRNA is a primary response to growth-factor stimulation of AKR-2B cells. Proc Natl Acad Sci U S A 85:1119-23