Human Susceptibility to Genetic Damage and Skin Cancer

O'Neill, James P.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Cancer Institute (NCI)
• Type: Research Project (R01)
• Project #: 5R01CA068288-02
• Application #: 2112207
• Study Section: Special Emphasis Panel (SRC (09))
• Project Start: 1995-09-01
• Project End: 1999-06-30
• Budget Start: 1996-07-01
• Budget End: 1997-06-30
• Support Year: 2
• Fiscal Year: 1996

Institution

• Name: University of Vermont & St Agric College
• Department: Internal Medicine/Medicine
• Type: Schools of Medicine
• DUNS #: 066811191
• City: Burlington
• State: VT
• Country: United States
• Zip Code: 05405

Publications

O'Neill, J P (2000) DNA damage, DNA repair, cell proliferation, and DNA replication: how do gene mutations result? Proc Natl Acad Sci U S A 97:11137-9

O'Neill, J P; Finette, B A (1998) Transition mutations at CpG dinucleotides are the most frequent in vivo spontaneous single-based substitution mutation in the human HPRT gene. Environ Mol Mutagen 32:188-91

Gundel, R M; O'Neill, J P (1997) Metabolic cooperation in the selection of thioguanine-resistant mutants does not occur with the human B-lymphoblastoid cell TK6. Environ Mol Mutagen 30:359-61