Mechanism of Ras in Tumor Progression - Bin-Tao Pan

Abstract

Funding Agency

Agency: National Institute of Health (NIH)
Institute: National Cancer Institute (NCI)
Type: Research Project (R01)
Project #: 5R01CA069282-05
Application #: 6376198
Study Section: Metabolic Pathology Study Section (MEP)

Project Start: 1996-05-01
Project End: 2004-05-31
Budget Start: 2001-06-01
Budget End: 2002-05-31
Support Year: 5
Fiscal Year: 2001
Total Cost: $206,364
Indirect Cost

Institution

Name: University of Kentucky
Department: Surgery
Type: Schools of Medicine
DUNS #: 832127323

City: Lexington
State: KY
Country: United States
Zip Code: 40506

Related projects


NIH 2003 R01 CA	Mechanism of Ras in Tumor Progression Pan, Bin-Tao B. / University of Kentucky	$206,340
NIH 2002 R01 CA	Mechanism of Ras in Tumor Progression Pan, Bin-Tao B. / University of Kentucky	$206,340
NIH 2001 R01 CA	Mechanism of Ras in Tumor Progression Pan, Bin-Tao B. / University of Kentucky	$206,364
NIH 2000 R01 CA	Mechanism of Ras in Tumor Progression Pan, Bin-Tao B. / University of Kentucky	$230,375
NIH 1998 R01 CA	Mechanism of Ras in Tumor Progression Pan, Bin-Tao B. / University of Kentucky
NIH 1997 R01 CA	Mechanism of Ras in Tumor Progression Pan, Bin-Tao B. / University of Kentucky
NIH 1996 R01 CA	Mechanism of Ras in Tumor Progression Pan, Bin-Tao B. / University of Kentucky

Publications

Huang, Tun-Lan; Pian, Jerry P; Pan, Bin-Tao (2013) Oncogenic Ras suppresses Cdk1 in a complex manner during the incubation of activated Xenopus egg extracts. Arch Biochem Biophys 532:61-72

Pian, Jerry Pinghwa; Huang, Tun-Lan; Tsai, Pei-Chi et al. (2004) A 32 kDa protein--whose phosphorylation correlates with oncogenic Ras-induced cell cycle arrest in activated Xenopus egg extracts--is identified as ribosomal protein S6. J Cell Physiol 201:305-19

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