Premature Cellular Senescence and the Lung

Adami, Guy R.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Cancer Institute (NCI)
• Type: Research Project (R01)
• Project #: 5R01CA085529-04
• Application #: 6633654
• Study Section: Metabolic Pathology Study Section (MEP)
• Program Officer: Forry, Suzanne L
• Project Start: 2000-07-07
• Project End: 2005-06-30
• Budget Start: 2003-07-01
• Budget End: 2004-06-30
• Support Year: 4
• Fiscal Year: 2003
• Total Cost: $174,713

Institution

• Name: University of Illinois at Chicago
• Department: Anatomy/Cell Biology
• Type: Schools of Medicine
• DUNS #: 098987217
• City: Chicago
• State: IL
• Country: United States
• Zip Code: 60612

Publications

Baranski, O A; Kalinichenko, V V; Adami, G R (2015) Increased FOXM1 expression can stimulate DNA repair in normal hepatocytes in vivo but also increases nuclear foci associated with senescence. Cell Prolif 48:105-15

Schwartz, Joel L; Panda, Suchismita; Beam, Craig et al. (2008) RNA from brush oral cytology to measure squamous cell carcinoma gene expression. J Oral Pathol Med 37:70-7

Panda, Suchismita; Isbatan, Ayman; Adami, Guy R (2008) Modification of the ATM/ATR directed DNA damage response state with aging and long after hepatocyte senescence induction in vivo. Mech Ageing Dev 129:332-40

Kalinina, Olga A; Kalinin, Sergey A; Polack, Evelyne W et al. (2003) Sustained hepatic expression of FoxM1B in transgenic mice has minimal effects on hepatocellular carcinoma development but increases cell proliferation rates in preneoplastic and early neoplastic lesions. Oncogene 22:6266-76