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Huang, Yufeng; Border, Wayne A; Lawrence, Daniel A et al. (2009) Mechanisms underlying the antifibrotic properties of noninhibitory PAI-1 (PAI-1R) in experimental nephritis. Am J Physiol Renal Physiol 297:F1045-54
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Huang, Y; Border, W A; Lawrence, D A et al. (2006) Noninhibitory PAI-1 enhances plasmin-mediated matrix degradation both in vitro and in experimental nephritis. Kidney Int 70:515-22
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Huang, Y; Wongamorntham, S; Kasting, J et al. (2006) Renin increases mesangial cell transforming growth factor-beta1 and matrix proteins through receptor-mediated, angiotensin II-independent mechanisms. Kidney Int 69:105-13
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Gaedeke, Jens; Noble, Nancy A; Border, Wayne A (2005) Curcumin blocks fibrosis in anti-Thy 1 glomerulonephritis through up-regulation of heme oxygenase 1. Kidney Int 68:2042-9
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Yu, Ling; Border, Wayne A; Anderson, Ian et al. (2004) Combining TGF-beta inhibition and angiotensin II blockade results in enhanced antifibrotic effect. Kidney Int 66:1774-84
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Peters, Harm; Border, Wayne A; Ruckert, Matthias et al. (2003) L-arginine supplementation accelerates renal fibrosis and shortens life span in experimental lupus nephritis. Kidney Int 63:1382-92
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Huang, Yufeng; Haraguchi, Masashi; Lawrence, Daniel A et al. (2003) A mutant, noninhibitory plasminogen activator inhibitor type 1 decreases matrix accumulation in experimental glomerulonephritis. J Clin Invest 112:379-88
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Yu, Ling; Border, Wayne A; Huang, Yufeng et al. (2003) TGF-beta isoforms in renal fibrogenesis. Kidney Int 64:844-56
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Haraguchi, M; Border, W A; Huang, Y et al. (2001) t-PA promotes glomerular plasmin generation and matrix degradation in experimental glomerulonephritis. Kidney Int 59:2146-55
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Border, W A; Noble, N (2001) Maximizing hemodynamic-independent effects of angiotensin II antagonists in fibrotic diseases. Semin Nephrol 21:563-72
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Showing the most recent 10 out of 25 publications
