Funding Agency

Agency
National Institute of Health (NIH)
Institute
National Institute of Diabetes and Digestive and Kidney Diseases (NIDDK)
Type
Research Project (R01)
Project #
5R01DK064169-02
Application #
6710585
Study Section
Metabolism Study Section (MET)
Program Officer
Malozowski, Saul N
Project Start
2003-03-01
Project End
2008-01-31
Budget Start
2004-02-01
Budget End
2005-01-31
Support Year
2
Fiscal Year
2004
Total Cost
$323,595
Indirect Cost

  Institution

Name
Cedars-Sinai Medical Center
Department
Type
DUNS #
075307785
City
Los Angeles
State
CA
Country
United States
Zip Code
90048

  Publications

Chesnokova, Vera; Wong, Chris; Zonis, Svetlana et al. (2009) Diminished pancreatic beta-cell mass in securin-null mice is caused by beta-cell apoptosis and senescence. Endocrinology 150:2603-10
Vlotides, George; Eigler, Tamar; Melmed, Shlomo (2007) Pituitary tumor-transforming gene: physiology and implications for tumorigenesis. Endocr Rev 28:165-86
Yu, Run; Cruz-Soto, Martha; Li Calzi, Sergio et al. (2006) Murine pituitary tumor-transforming gene functions as a securin protein in insulin-secreting cells. J Endocrinol 191:45-53
Lum, Pek Yee; Chen, Yanqing; Zhu, Jun et al. (2006) Elucidating the murine brain transcriptional network in a segregating mouse population to identify core functional modules for obesity and diabetes. J Neurochem 97 Suppl 1:50-62
Fraenkel, M; Caloyeras, J; Ren, S-G et al. (2006) Sex-steroid milieu determines diabetes rescue in pttg-null mice. J Endocrinol 189:519-28
Melmed, Shlomo (2003) Mechanisms for pituitary tumorigenesis: the plastic pituitary. J Clin Invest 112:1603-18