Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
Research Project (R01)
Project #
5R01GM032288-08
Application #
2176506
Study Section
Surgery, Anesthesiology and Trauma Study Section (SAT)
Project Start
1984-12-01
Project End
1997-08-31
Budget Start
1994-09-01
Budget End
1997-08-31
Support Year
8
Fiscal Year
1994
Total Cost
Indirect Cost
Name
Loyola University Chicago
Department
Physiology
Type
Schools of Medicine
DUNS #
791277940
City
Maywood
State
IL
Country
United States
Zip Code
60153
Choudhry, M A; Uddin, S; Sayeed, M M (1998) Prostaglandin E2 modulation of p59fyn tyrosine kinase in T lymphocytes during sepsis. J Immunol 160:929-35
Rose, S; Sayeed, M M (1997) Superoxide radical scavenging prevents cellular calcium dysregulation during intraabdominal sepsis. Shock 7:263-8
Fazal, N; Sabeh, F; Gamelli, R L et al. (1997) Elevated expression of p47phox and p67phox proteins in neutrophils from burned rats. Shock 8:256-60
Ahmad, S; Choudhry, M A; Shankar, R et al. (1997) Transforming growth factor-beta negatively modulates T-cell responses in sepsis. FEBS Lett 402:213-8
Sayeed, M M (1996) Alterations in calcium signaling and cellular responses in septic injury. New Horiz 4:72-86
Ahmad, S; Choudhry, M A; Sayeed, M M (1996) Ca2(+)-dependent and Ca2(+)-independent proteinase contents in the skeletal muscle in septic rats. Shock 5:247-50
Choudhry, M A; Sayeed, M M (1996) Calcium signaling restitution prevents T-cell proliferative suppression by prostaglandin E2. Shock 6:101-5
Sayeed, M M (1996) Alterations in cell signaling and related effector functions in T lymphocytes in burn/trauma/septic injuries. Shock 5:157-66
Choudhry, M A; Ahmad, S; Sayeed, M M (1995) Role of Ca2+ in prostaglandin E2-induced T-lymphocyte proliferative suppression in sepsis. Infect Immun 63:3101-5
Choudhry, M A; Ahmad, S; Thompson, K D et al. (1994) T-lymphocyte Ca2+ signalling and proliferative responses during sepsis. Shock 1:267-72

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