Agency
National Institute of Health (NIH)
Institute
National Institute of General Medical Sciences (NIGMS)
Type
Research Project (R01)
Project #
1R01GM042850-01
Application #
3898447
Study Section
Project Start
Project End
Budget Start
Budget End
Support Year
1
Fiscal Year
1989
Total Cost
Indirect Cost
Name
State University of New York at Buffalo
Department
Type
DUNS #
038633251
City
Buffalo
State
NY
Country
United States
Zip Code
14260
Fung, H L; Booth, B P; Tabrizi-Fard, M (1999) Therapeutics of nitric oxide modulation. Met Ions Biol Syst 36:723-49
Booth, B P; Fung, H L (1998) Contribution of vascular tissue to the antiplatelet activity of sodium nitroprusside. J Cardiovasc Pharmacol 32:129-33
Booth, B P; Nolan, T D; Fung, H L (1997) Nitroglycerin-inhibited whole blood aggregation is partially mediated by calcitonin gene-related peptide -- a neurogenic mechanism. Br J Pharmacol 122:577-83
Bauer, J A; Nolan, T; Fung, H L (1997) Vascular and hemodynamic differences between organic nitrates and nitrites. J Pharmacol Exp Ther 280:326-31
Bauer, J A; Balthasar, J P; Fung, H L (1997) Application of pharmacodynamic modeling for designing time-variant dosing regimens to overcome nitroglycerin tolerance in experimental heart failure. Pharm Res 14:1140-5
Booth, B P; Jacob, S; Bauer, J A et al. (1996) Sustained antiplatelet properties of nitroglycerin during hemodynamic tolerance in rats. J Cardiovasc Pharmacol 28:432-8
Bauer, J A; Booth, B P; Fung, H L (1995) Nitric oxide donors: biochemical pharmacology and therapeutics. Adv Pharmacol 34:361-81
Bangalore, R; Triggle, D J (1995) Age-dependent changes in voltage-gated calcium channels and ATP-dependent potassium channels in Fischer 344 rats. Gen Pharmacol 26:1237-42
Fung, H L; Bauer, J A (1994) Mechanisms of nitrate tolerance. Cardiovasc Drugs Ther 8:489-99
Bauer, J A; Fung, H L (1994) Pharmacodynamic models of nitroglycerin-induced hemodynamic tolerance in experimental heart failure. Pharm Res 11:816-23

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