Agency
National Institute of Health (NIH)
Institute
National Heart, Lung, and Blood Institute (NHLBI)
Type
Research Project (R01)
Project #
7R01HL038537-07
Application #
3354859
Study Section
Experimental Cardiovascular Sciences Study Section (ECS)
Project Start
1987-09-01
Project End
1993-08-31
Budget Start
1992-06-19
Budget End
1992-08-31
Support Year
7
Fiscal Year
1991
Total Cost
Indirect Cost
Name
Oregon Regional Primate Research Center
Department
Type
DUNS #
City
Beaverton
State
OR
Country
United States
Zip Code
97006
Hermsmeyer, K; White, A C; Triggle, D J (1995) Decreased dihydropyridine receptor number in hypertensive rat vascular muscle cells. Hypertension 25:731-4
Self, D A; Bian, K; Mishra, S K et al. (1994) Stroke-prone SHR vascular muscle Ca2+ current amplitudes correlate with lethal increases in blood pressure. J Vasc Res 31:359-66
Mishra, S K; Hermsmeyer, K (1994) Resting state block and use independence of rat vascular muscle Ca++ channels by Ro 40-5967. J Pharmacol Exp Ther 269:178-83
Mishra, S K; Hermsmeyer, K (1994) Selective inhibition of T-type Ca2+ channels by Ro 40-5967. Circ Res 75:144-8
Mishra, S K; Hermsmeyer, K (1994) Inhibition of signal Ca2+ in dog coronary arterial vascular muscle cells by Ro 40-5967. J Cardiovasc Pharmacol 24:1-7
Bian, K; Hermsmeyer, K (1994) Glyburide actions on the dihydropyridine-sensitive Ca2+ channel in rat vascular muscle. J Vasc Res 31:256-64
Bian, K; Hermsmeyer, K (1993) Ca2+ channel actions of the non-dihydropyridine Ca2+ channel antagonist Ro 40-5967 in vascular muscle cells cultured from dog coronary and saphenous arteries. Naunyn Schmiedebergs Arch Pharmacol 348:191-6
Hermsmeyer, K (1993) Calcium channel function in hypertension. J Hum Hypertens 7:173-6
Baker, J R; Hedwall, P R; Hermsmeyer, K (1992) Subcellular distribution of hydralazine in rat single vascular muscle cells. Cell Biol Int Rep 16:1023-39
Erne, P; Hermsmeyer, K (1991) Modulation of intracellular calcium by potassium channel openers in vascular muscle. Naunyn Schmiedebergs Arch Pharmacol 344:706-15

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