Mechanisms of Endothelial Cell Fibrinolysis in Blacks

Booyse, Francois M.

Abstract

Funding Agency

• Agency: National Institute of Health (NIH)
• Institute: National Heart, Lung, and Blood Institute (NHLBI)
• Type: Research Project (R01)
• Project #: 5R01HL049764-05
• Application #: 2028859
• Study Section: Special Emphasis Panel (ZHL1-CSR-K (01))
• Project Start: 1992-09-30
• Project End: 1998-04-30
• Budget Start: 1996-09-01
• Budget End: 1998-04-30
• Support Year: 5
• Fiscal Year: 1996

Institution

• Name: University of Alabama Birmingham
• Department: Internal Medicine/Medicine
• Type: Schools of Medicine
• DUNS #: 004514360
• City: Birmingham
• State: AL
• Country: United States
• Zip Code: 35294

Publications

Grenett, H E; Benza, R L; Li, X N et al. (1999) Expression of plasminogen activator inhibitor type I in genotyped human endothelial cell cultures: genotype-specific regulation by insulin. Thromb Haemost 82:1504-9

Benza, R L; Grenett, H E; Bourge, R C et al. (1998) Gene polymorphisms for plasminogen activator inhibitor-1/tissue plasminogen activator and development of allograft coronary artery disease. Circulation 98:2248-54

Li, X N; Varma, V K; Parks, J M et al. (1995) Thrombin decreases the urokinase receptor and surface-localized fibrinolysis in cultured endothelial cells. Arterioscler Thromb Vasc Biol 15:410-9

Frist, S T; Taylor Jr, H A; Kirk, K A et al. (1995) Expression of PAI-1, t-PA and u-PA in cultured human umbilical vein endothelial cells derived from racial groups. Thromb Res 77:279-90